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Reinhard Wallich

Name	Reinhard Wallich
Ort	Deutschland
Geschlecht	männlich
Akademischer Grad bzw. Titel	apl. Prof. Dr. rer. nat.
Beziehung	Universität Heidelberg. Institut für Immunologie und Serologie
Weitere Angaben	Institut für Immunologie, Heidelberg, Abteilung Allgemeine Immunologie, Serologie und Hämatologische Diagnostik mit Blutbank
Datensatztyp	Person
GND-Nummer	1059566710
Datensatz-ID	1059566710
Beteiligt an alle anzeigen Author Correction: Immune evasion of Borrelia miyamotoi: CbiA, a novel outer surface protein exhibiting complement binding and inactivating properties Immune evasion of Borrelia miyamotoi: CbiA, a novel outer surface protein exhibiting complement binding and inactivating properties First insights in the variability of Borrelia recurrentis genomes Immune evasion of Borrelia miyamotoi : CbiA, a novel outer surface protein exhibiting complement binding and inactivating properties Identification and characterization of a unique role for EDB fibronectin in phagocytosis Identification and characterization of a unique role for EDB fibronectin in phagocytosis Whole-chain tick saliva proteins presented on hepatitis B virus capsid-like particles induce high-titered antibodies with neutralizing potential Borrelia valaisiana resist complement-mediated killing independently of the recruitment of immune regulators and inactivation of complement components Contribution of the infection-associated complement regulator-acquiring surface protein 4 (ErpC) to complement resistance of Borrelia burgdorferi Complement factor H-related proteins CFHR2 and CFHR5 represent novel ligands for the infection-associated CRASP proteins of Borrelia burgdorferi Borrelia recurrentis employs a novel multifunctional surface protein with anti-complement, anti-opsonic and invasive potential to escape innate immunity Resolution of experimental and tick‐borne Borrelia burgdorferi infection in mice by passive, but not active immunization using recombinant OspC A fusion product of the complete Borrelia burgdorferi outer surface protein A (OspA) and the hepatitis B virus capsid protein is highly immunogenic and induces protective immunity similar to that seen with an effective lipidated OspA vaccine formula Direct Toll-like receptor 2 mediated co-stimulation of T cells in the mouse system as a basis for chronic inflammatory joint disease Quantitative analysis of Borrelia burgdorferi gene expression in naturally (tick) infected mouse strains Artificial‐infection protocols allow immunodetection of novel Borrelia burgdorferi antigens suitable as vaccine candidates against Lyme disease Prominent T cell response to a selectively in vivo expressed Borrelia burgdorferi outer surface protein (pG) in patients with Lyme disease Änderung der Malignität eines Maustumors durch Transfektion mit klonierten Histokompatibilitätsgenen

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Reinhard Wallich

Author Correction: Immune evasion of Borrelia miyamotoi: CbiA, a novel outer surface protein exhibiting complement binding and inactivating properties

Immune evasion of Borrelia miyamotoi: CbiA, a novel outer surface protein exhibiting complement binding and inactivating properties

First insights in the variability of Borrelia recurrentis genomes

Immune evasion of Borrelia miyamotoi : CbiA, a novel outer surface protein exhibiting complement binding and inactivating properties

Identification and characterization of a unique role for EDB fibronectin in phagocytosis

Identification and characterization of a unique role for EDB fibronectin in phagocytosis

Whole-chain tick saliva proteins presented on hepatitis B virus capsid-like particles induce high-titered antibodies with neutralizing potential

Borrelia valaisiana resist complement-mediated killing independently of the recruitment of immune regulators and inactivation of complement components

Contribution of the infection-associated complement regulator-acquiring surface protein 4 (ErpC) to complement resistance of Borrelia burgdorferi

Complement factor H-related proteins CFHR2 and CFHR5 represent novel ligands for the infection-associated CRASP proteins of Borrelia burgdorferi

Borrelia recurrentis employs a novel multifunctional surface protein with anti-complement, anti-opsonic and invasive potential to escape innate immunity

Resolution of experimental and tick‐borne Borrelia burgdorferi infection in mice by passive, but not active immunization using recombinant OspC

A fusion product of the complete Borrelia burgdorferi outer surface protein A (OspA) and the hepatitis B virus capsid protein is highly immunogenic and induces protective immunity similar to that seen with an effective lipidated OspA vaccine formula

Direct Toll-like receptor 2 mediated co-stimulation of T cells in the mouse system as a basis for chronic inflammatory joint disease

Quantitative analysis of Borrelia burgdorferi gene expression in naturally (tick) infected mouse strains

Artificial‐infection protocols allow immunodetection of novel Borrelia burgdorferi antigens suitable as vaccine candidates against Lyme disease

Prominent T cell response to a selectively in vivo expressed Borrelia burgdorferi outer surface protein (pG) in patients with Lyme disease

Änderung der Malignität eines Maustumors durch Transfektion mit klonierten Histokompatibilitätsgenen