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Wilhelm Bertrams

Biologe

Name	Wilhelm Bertrams
Ort	Deutschland
Geschlecht	männlich
Akademischer Grad bzw. Titel	Dr. rer. nat.
Beruf	Biologe (charakteristischer Beruf) Molekularbiologe
Variante	Wilhelm Goswin Bertrams
Beziehung	Universitätsklinikum Gießen und Marburg. Standort Marburg Universities of Giessen and Marburg Lung Center
Weitere Angaben	Diss. Humboldt-Univ. Berlin 2014
Datensatztyp	Person
GND-Nummer	1064676766
Datensatz-ID	1064676766
Beteiligt an alle anzeigen A mRNA panel for differentiation between acute exacerbation or pneumonia in COPD patients Eosinopenia as predictor of disease severity in patients with community-acquired pneumonia: an observational study Cell-free biosynthesis combined with deep learning accelerates de novo-development of antimicrobial peptides NAD+ metabolism is a key modulator of bacterial respiratory epithelial infections Streptococcus pneumoniae disrupts the structure of the golgi apparatus and subsequent epithelial cytokine response in an H2O2-dependent manner Bacterial vesicles block viral replication in macrophages via TLR4-TRIF-axis Surface proteome of plasma extracellular vesicles as mechanistic and clinical biomarkers for malaria Pulmonary inflammatory response and immunomodulation to multiple trauma and hemorrhagic shock in pigs Screening for eukaryotic motifs in Legionella pneumophila reveals Smh1 as bacterial deacetylase of host histones Transcriptomic comparison of primary human lung cells with lung tissue samples and the human A549 lung cell line highlights cell type specific responses during infections with influenza A virus Lactate induces metabolic and epigenetic reprogramming of pro‐inflammatory Th17 cells Selected commensals educate the intestinal vascular and immune system for immunocompetence ADAM8 signaling drives neutrophil migration and ARDS severity Bedeutung der Genetik für die Pneumonie und COVID-19 Intracellular bacteria engage a STING–TBK1–MVB12b pathway to enable paracrine cGAS–STING signalling microRNA-125a-3p is regulated by MyD88 in Legionella pneumophila infection and targets NTAN1 MicroRNAs in alternative and classic activation of macrophages

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Katalog der Deutschen Nationalbibliothek

Wilhelm Bertrams

A mRNA panel for differentiation between acute exacerbation or pneumonia in COPD patients

Eosinopenia as predictor of disease severity in patients with community-acquired pneumonia: an observational study

Cell-free biosynthesis combined with deep learning accelerates de novo-development of antimicrobial peptides

NAD+ metabolism is a key modulator of bacterial respiratory epithelial infections

Streptococcus pneumoniae disrupts the structure of the golgi apparatus and subsequent epithelial cytokine response in an H2O2-dependent manner

Bacterial vesicles block viral replication in macrophages via TLR4-TRIF-axis

Surface proteome of plasma extracellular vesicles as mechanistic and clinical biomarkers for malaria

Pulmonary inflammatory response and immunomodulation to multiple trauma and hemorrhagic shock in pigs

Screening for eukaryotic motifs in Legionella pneumophila reveals Smh1 as bacterial deacetylase of host histones

Transcriptomic comparison of primary human lung cells with lung tissue samples and the human A549 lung cell line highlights cell type specific responses during infections with influenza A virus

Lactate induces metabolic and epigenetic reprogramming of pro‐inflammatory Th17 cells

Selected commensals educate the intestinal vascular and immune system for immunocompetence

ADAM8 signaling drives neutrophil migration and ARDS severity

Bedeutung der Genetik für die Pneumonie und COVID-19

Intracellular bacteria engage a STING–TBK1–MVB12b pathway to enable paracrine cGAS–STING signalling

microRNA-125a-3p is regulated by MyD88 in Legionella pneumophila infection and targets NTAN1

MicroRNAs in alternative and classic activation of macrophages