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Ian Frew (1976-)

Physiologe

Name	Ian Frew
Zeit	1976- (Lebensdaten)
Ort	Sonstiges Ausland / Unbekanntes Land
Beruf	Physiologe (charakteristischer Beruf)
Variante	Ian J. Frew
Weitere Angaben	Physiologe
Datensatztyp	Person
GND-Nummer	1089675941
Datensatz-ID	1089675941
Beteiligt an alle anzeigen Nonstop mutations cause loss of renal tumor suppressor proteins VHL and BAP1 and affect multiple stages of protein translation Characterization of the immunosuppressive tumour microenvironment in a mouse model of clear cell renal cell carcinoma Integrated metabolomic and transcriptomic analysis of modified nucleosides for biomarker discovery in clear cell renal cell carcinoma SCD5 regulation by VHL affects cell proliferation and lipid homeostasis in ccRCC The secretome of colorectal cancer influences metastasis and the tumor microenvironment modulates its dysregulation ATR represents a therapeutic vulnerability in clear cell renal cell carcinoma MARVEL-domain containing CMTM4 affects CXCR4 trafficking Noninvasive CNS lymphoma risk stratification and brain cancer classification by circulating tumor DNA profiling The role of PBRM1 in clear cell renal cell carcinoma Therapeutic significance of ARID1A mutation in bladder cancer Sensitivity and resistance of oncogenic RAS-driven tumors to dual MEK and ERK inhibition Sensitivity and resistance of oncogenic RAS-driven tumors to dual MEK and ERK inhibition Therapeutic effects of inhibition of sphingosine-1-phosphate signaling in HIF-2[alpha] inhibitor-resistant clear cell renal cell carcinoma VHL suppresses RAPTOR and inhibits mTORC1 signaling in clear cell renal cell carcinoma The complex genetics of epigenetics in urothelial carcinomas HIF-1[alpha] and HIF-2[alpha] differently regulate tumour development and inflammation of clear cell renal cell carcinoma in mice IL‐8 and CXCR1 expression is associated with cancer stem cell‐like properties of clear cell renal cancer Mouse genetic background influences whether Hras(G12V) expression plus Cdkn2a knockdown causes angiosarcoma or undifferentiated pleomorphic sarcoma Evidence of renal angiomyolipoma neoplastic stem cells arising from renal epithelial cells Evidence of renal angiomyolipoma neoplastic stem cells arising from renal epithelial cells Oncogenic HrasG12V expression plus knockdown of Cdkn2a using ecotropic lentiviral vectors induces high-grade endometrial stromal sarcoma

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Ian Frew (1976-)

Nonstop mutations cause loss of renal tumor suppressor proteins VHL and BAP1 and affect multiple stages of protein translation

Characterization of the immunosuppressive tumour microenvironment in a mouse model of clear cell renal cell carcinoma

Integrated metabolomic and transcriptomic analysis of modified nucleosides for biomarker discovery in clear cell renal cell carcinoma

SCD5 regulation by VHL affects cell proliferation and lipid homeostasis in ccRCC

The secretome of colorectal cancer influences metastasis and the tumor microenvironment modulates its dysregulation

ATR represents a therapeutic vulnerability in clear cell renal cell carcinoma

MARVEL-domain containing CMTM4 affects CXCR4 trafficking

Noninvasive CNS lymphoma risk stratification and brain cancer classification by circulating tumor DNA profiling

The role of PBRM1 in clear cell renal cell carcinoma

Therapeutic significance of ARID1A mutation in bladder cancer

Sensitivity and resistance of oncogenic RAS-driven tumors to dual MEK and ERK inhibition

Sensitivity and resistance of oncogenic RAS-driven tumors to dual MEK and ERK inhibition

Therapeutic effects of inhibition of sphingosine-1-phosphate signaling in HIF-2[alpha] inhibitor-resistant clear cell renal cell carcinoma

VHL suppresses RAPTOR and inhibits mTORC1 signaling in clear cell renal cell carcinoma

The complex genetics of epigenetics in urothelial carcinomas

HIF-1[alpha] and HIF-2[alpha] differently regulate tumour development and inflammation of clear cell renal cell carcinoma in mice

IL‐8 and CXCR1 expression is associated with cancer stem cell‐like properties of clear cell renal cancer

Mouse genetic background influences whether Hras(G12V) expression plus Cdkn2a knockdown causes angiosarcoma or undifferentiated pleomorphic sarcoma

Evidence of renal angiomyolipoma neoplastic stem cells arising from renal epithelial cells

Evidence of renal angiomyolipoma neoplastic stem cells arising from renal epithelial cells

Oncogenic HrasG12V expression plus knockdown of Cdkn2a using ecotropic lentiviral vectors induces high-grade endometrial stromal sarcoma