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Ashish Goyal

Name	Ashish Goyal
Ort	Jaipur (Geburtsort) Indien
Akademischer Grad bzw. Titel	Dr. rer. nat.
Beziehung	Deutsches Krebsforschungszentrum Universität Heidelberg. Pathologisches Institut (2016-2017)
Weitere Angaben	Division of RNA Biology & Cancer, German Cancer Research Center (DKFZ), Institute of Pathology, University Hospital Heidelberg
Datensatztyp	Person
GND-Nummer	113560018X
Datensatz-ID	113560018X
Beteiligt an alle anzeigen DNMT and HDAC inhibition induces immunogenic neoantigens from human endogenous retroviral element-derived transcripts DNMT and HDAC inhibition induces immunogenic neoantigens from human endogenous retroviral element-derived transcripts Metal cutting processes Metal Cutting Processes Metal Cutting Processes Epigenetic modulation of radiation-induced diacylglycerol kinase alpha expression prevents pro-fibrotic fibroblast response A cautionary tale of sense-antisense gene pairs: independent regulation despite inverse correlation of expression CRISPR/Cas9-mediated interrogation of a sense-antisense pair involved in the DNA damage response CRISPR/Cas9-mediated interrogation of a sense-antisense pair involved in the DNA damage response Generation of murine tumor cell lines deficient in MHC molecule surface expression using the CRISPR/Cas9 system Challenges of CRISPR/Cas9 applications for long non-coding RNA genes

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Ashish Goyal

DNMT and HDAC inhibition induces immunogenic neoantigens from human endogenous retroviral element-derived transcripts

DNMT and HDAC inhibition induces immunogenic neoantigens from human endogenous retroviral element-derived transcripts

Metal cutting processes

Metal Cutting Processes

Metal Cutting Processes

Epigenetic modulation of radiation-induced diacylglycerol kinase alpha expression prevents pro-fibrotic fibroblast response

A cautionary tale of sense-antisense gene pairs: independent regulation despite inverse correlation of expression

CRISPR/Cas9-mediated interrogation of a sense-antisense pair involved in the DNA damage response

CRISPR/Cas9-mediated interrogation of a sense-antisense pair involved in the DNA damage response

Generation of murine tumor cell lines deficient in MHC molecule surface expression using the CRISPR/Cas9 system

Challenges of CRISPR/Cas9 applications for long non-coding RNA genes