Detailansicht

Wissensnetz

Person

Brian A. Walker

Arzt

Name	Brian A. Walker
Ort	USA
Geschlecht	männlich
Akademischer Grad bzw. Titel	Prof.
Beruf	Arzt (charakteristischer Beruf)
Beziehung	University of Arkansas for Medical Sciences
Weitere Angaben	Myeloma Institute, University of Arkansas for Medical Sciences, Little Rock, AR
Datensatztyp	Person
GND-Nummer	1184512817
Datensatz-ID	1184512817
Beteiligt an alle anzeigen 1q amplification and PHF19 expressing high-risk cells are associated with relapsed/refractory multiple myeloma A high-risk, Double-Hit, group of newly diagnosed myeloma identified by genomic analysis Identifying novel mechanisms of biallelic TP53 loss refines poor outcome for patients with multiple myeloma The spatio-temporal evolution of multiple myeloma from baseline to relapse-refractory states The molecular make up of smoldering myeloma highlights the evolutionary pathways leading to multiple myeloma The functional epigenetic landscape of aberrant gene expression in molecular subgroups of newly diagnosed multiple myeloma Transcriptome-wide association study of multiple myeloma identifies candidate susceptibility genes Combination of flow cytometry and functional imaging for monitoring of residual disease in myeloma A high-risk, Double-Hit, group of newly diagnosed myeloma identified by genomic analysis Kinase domain activation through gene rearrangement in multiple myeloma Loss of heterozygosity as a marker of homologous repair deficiency in multiple myeloma: a role for PARP inhibition?

Hinweis

Die Betaversion beinhaltet noch nicht alle Funktionen und Informationen des Katalogs des DNB-Portals. Falls Sie Informationen vermissen oder ein Medium bestellen wollen, besuchen Sie bitte die entsprechende Seite im Katalog des DNB-Portals über folgenden Link:

Katalog der Deutschen Nationalbibliothek

Brian A. Walker

1q amplification and PHF19 expressing high-risk cells are associated with relapsed/refractory multiple myeloma

A high-risk, Double-Hit, group of newly diagnosed myeloma identified by genomic analysis

Identifying novel mechanisms of biallelic TP53 loss refines poor outcome for patients with multiple myeloma

The spatio-temporal evolution of multiple myeloma from baseline to relapse-refractory states

The molecular make up of smoldering myeloma highlights the evolutionary pathways leading to multiple myeloma

The functional epigenetic landscape of aberrant gene expression in molecular subgroups of newly diagnosed multiple myeloma

Transcriptome-wide association study of multiple myeloma identifies candidate susceptibility genes

Combination of flow cytometry and functional imaging for monitoring of residual disease in myeloma

A high-risk, Double-Hit, group of newly diagnosed myeloma identified by genomic analysis

Kinase domain activation through gene rearrangement in multiple myeloma

Loss of heterozygosity as a marker of homologous repair deficiency in multiple myeloma: a role for PARP inhibition?