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Carmen Härdtner

Wissenschaftliche Mitarbeiterin

Name	Carmen Härdtner
Ort	Deutschland
Geschlecht	weiblich
Akademischer Grad bzw. Titel	Dr. rer. nat.
Beruf	Wissenschaftliche Mitarbeiterin (charakteristischer Beruf)
Beziehung	Biochemie (Studienfach) Molekularbiologie (Studienfach) Cytologie (Studienfach)
Weitere Angaben	Postdoc beim Universitäts-Herzzentrum Freiburg Bad Krozingen
Quelle	FreiDok plus
Datensatztyp	Person
GND-Nummer	1185194983
Datensatz-ID	1185194983
Beteiligt an alle anzeigen A comparative gene expression matrix in Apoe-deficient mice identifies unique and atherosclerotic disease stage-specific gene regulation patterns in monocytes and macrophages Elevated platelet–leukocyte complexes are associated with, but dispensable for myocardial ischemia–reperfusion injury GPR55 in B cells limits atherosclerosis development and regulates plasma cell maturation Der Einfluss des Cholesterinexports über ATP-binding cassette Transporter auf das Makrophagenverhalten in der atherosklerotischen Plaque = Influence of ABC-transporter mediated cholesterol export on macrophage turnover in the atherosclerotic plaque Elevated platelet–leukocyte complexes are associated with, but dispensable for myocardial ischemia–reperfusion injury Everolimus-loaded reconstituted high-density lipoprotein prepared by a novel dual centrifugation approach for Anti-atherosclerotic therapy GPR55 in B cells limits atherosclerosis development and regulates plasma cell maturation P2X4 deficiency reduces atherosclerosis and plaque inflammation in mice Myeloid cell-specific Irf5 deficiency stabilizes atherosclerotic plaques in Apoe–/– mice The role of interferon regulatory factor 8 for retinal tissue homeostasis and development of choroidal neovascularisation The role of interferon regulatory factor 8 for retinal tissue homeostasis and development of choroidal neovascularisation Inhibition of macrophage proliferation dominates plaque regression in response to cholesterol lowering Inhibition of macrophage proliferation dominates plaque regression in response to cholesterol lowering Glucose lowering by SGLT2-inhibitor empagliflozin accelerates atherosclerosis regression in hyperglycemic STZ-diabetic mice Atheroprotection through SYK inhibition fails in established disease when local macrophage proliferation dominates lesion progression Atheroprotection through SYK inhibition fails in established disease when local macrophage proliferation dominates lesion progression

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Carmen Härdtner

A comparative gene expression matrix in Apoe-deficient mice identifies unique and atherosclerotic disease stage-specific gene regulation patterns in monocytes and macrophages

Elevated platelet–leukocyte complexes are associated with, but dispensable for myocardial ischemia–reperfusion injury

GPR55 in B cells limits atherosclerosis development and regulates plasma cell maturation

Der Einfluss des Cholesterinexports über ATP-binding cassette Transporter auf das Makrophagenverhalten in der atherosklerotischen Plaque = Influence of ABC-transporter mediated cholesterol export on macrophage turnover in the atherosclerotic plaque

Elevated platelet–leukocyte complexes are associated with, but dispensable for myocardial ischemia–reperfusion injury

Everolimus-loaded reconstituted high-density lipoprotein prepared by a novel dual centrifugation approach for Anti-atherosclerotic therapy

GPR55 in B cells limits atherosclerosis development and regulates plasma cell maturation

P2X4 deficiency reduces atherosclerosis and plaque inflammation in mice

Myeloid cell-specific Irf5 deficiency stabilizes atherosclerotic plaques in Apoe–/– mice

The role of interferon regulatory factor 8 for retinal tissue homeostasis and development of choroidal neovascularisation

The role of interferon regulatory factor 8 for retinal tissue homeostasis and development of choroidal neovascularisation

Inhibition of macrophage proliferation dominates plaque regression in response to cholesterol lowering

Inhibition of macrophage proliferation dominates plaque regression in response to cholesterol lowering

Glucose lowering by SGLT2-inhibitor empagliflozin accelerates atherosclerosis regression in hyperglycemic STZ-diabetic mice

Atheroprotection through SYK inhibition fails in established disease when local macrophage proliferation dominates lesion progression

Atheroprotection through SYK inhibition fails in established disease when local macrophage proliferation dominates lesion progression