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Dennis W. Dickson

Arzt

Name	Dennis W. Dickson
Ort	USA
Geschlecht	männlich
Beruf	Arzt (charakteristischer Beruf) Hochschullehrer
Beziehung	Mayo Clinic
Weitere Angaben	Professor of Pathology, Departments of Pathology (Neuropathology) and Neuroscience, Mayo Clinic, Jacksonville, FL, USA
Quelle	https://www.mayo.edu/research/faculty/dickson-dennis-w-m-d/bio-00027499 (Stand: 06.05.2020)
Datensatztyp	Person
GND-Nummer	1209611597
Datensatz-ID	1209611597
Beteiligt an alle anzeigen Author Correction: Genetic, transcriptomic, histological, and biochemical analysis of progressive supranuclear palsy implicates glial activation and novel risk genes Author Correction: Divergent single cell transcriptome and epigenome alterations in ALS and FTD patients with C9orf72 mutation Genetic, transcriptomic, histological, and biochemical analysis of progressive supranuclear palsy implicates glial activation and novel risk genes Correction: HDGFL2 cryptic proteins report presence of TDP-43 pathology in neurodegenerative diseases Gliovascular transcriptional perturbations in Alzheimer’s disease reveal molecular mechanisms of blood brain barrier dysfunction Abundant transcriptomic alterations in the human cerebellum of patients with a C9orf72 repeat expansion Rare genetic variation in fibronectin 1 (FN1) protects against APOEε4 in Alzheimer’s disease A multimodal clinical diagnostic approach using MRI and 18F-FDG-PET for antemortem diagnosis of TDP-43 in cases with low–intermediate Alzheimer’s disease neuropathologic changes and primary age-related tauopathy Influences of amyloid-β and tau on white matter neurite alterations in dementia with Lewy bodies A systematic review of progranulin concentrations in biofluids in over 7,000 people—assessing the pathogenicity of GRN mutations and other influencing factors HDGFL2 cryptic proteins report presence of TDP-43 pathology in neurodegenerative diseases Role of GBA variants in Lewy body disease neuropathology Impact of APOE on amyloid and tau accumulation in argyrophilic grain disease and Alzheimer’s disease Cryptic splicing of stathmin-2 and UNC13A mRNAs is a pathological hallmark of TDP-43-associated Alzheimer’s disease Letter to the editor on: Hornerin deposits in neuronal intranuclear inclusion disease: direct identification of proteins with compositionally biased regions in inclusions by Park et al. (2022) Cross species systems biology discovers glial DDR2, STOM, and KANK2 as therapeutic targets in progressive supranuclear palsy Divergent single cell transcriptome and epigenome alterations in ALS and FTD patients with C9orf72 mutation TDP-43-regulated cryptic RNAs accumulate in Alzheimer’s disease brains Publisher Correction to: Diffuse argyrophilic grain disease with TDP-43 proteinopathy and neuronal intermediate filament inclusion disease: FTLD with mixed tau, TDP-43 and FUS pathologies Diffuse argyrophilic grain disease with TDP-43 proteinopathy and neuronal intermediate filament inclusion disease: FTLD with mixed tau, TDP-43 and FUS pathologies Case report of a patient with unclassified tauopathy with molecular and neuropathological features of both progressive supranuclear palsy and corticobasal degeneration Cancer in pathologically confirmed multiple system atrophy Genome-wide association study of brain biochemical phenotypes reveals distinct genetic architecture of Alzheimer’s disease related proteins Molecular fragment characteristics and distribution of tangle associated TDP-43 (TATs) and other TDP-43 lesions in Alzheimer’s disease Global neuropathologic severity of Alzheimer’s disease and locus coeruleus vulnerability influences plasma phosphorylated tau levels LATE-NC staging in routine neuropathologic diagnosis: an update Nuclear import receptors are recruited by FG-nucleoporins to rescue hallmarks of TDP-43 proteinopathy Structure-based discovery of small molecules that disaggregate Alzheimer’s disease tissue derived tau fibrils in vitro Discrepancy between distribution of alpha-synuclein oligomers and Lewy-related pathology in Parkinson’s disease Rainwater Charitable Foundation criteria for the neuropathologic diagnosis of progressive supranuclear palsy Evidence of cerebellar TDP-43 loss of function in FTLD-TDP Neuropathologic scales of cerebrovascular disease associated with diffusion changes on MRI Mitochondrial genomic variation in dementia with Lewy bodies: association with disease risk and neuropathological measures Frequency of LATE neuropathologic change across the spectrum of Alzheimer’s disease neuropathology: combined data from 13 community-based or population-based autopsy cohorts Frequency and distribution of TAR DNA-binding protein 43 (TDP-43) pathology increase linearly with age in a large cohort of older adults with and without dementia Clinical and pathological characteristics of later onset multiple system atrophy Proximity proteomics of C9orf72 dipeptide repeat proteins identifies molecular chaperones as modifiers of poly-GA aggregation TDP-43 Pathology in Alzheimer’s Disease Neuropathology and molecular diagnosis of Synucleinopathies Correction to: Apolipoprotein E regulates lipid metabolism and α‑synuclein pathology in human iPSC‑derived cerebral organoids Genome-wide association study and functional validation implicates JADE1 in tauopathy Apolipoprotein E regulates lipid metabolism and α-synuclein pathology in human iPSC-derived cerebral organoids Cellular and pathological heterogeneity of primary tauopathies Predictors of cognitive impairment in primary age-related tauopathy: an autopsy study Genome-wide analysis identifies a novel LINC-PINT splice variant associated with vascular amyloid pathology in Alzheimer’s disease Tau isoforms are differentially expressed across the hippocampus in chronic traumatic encephalopathy and Alzheimer’s disease Latent trait modeling of tau neuropathology in progressive supranuclear palsy Frequency of spinocerebellar ataxia mutations in patients with multiple system atrophy MAPT subhaplotypes in corticobasal degeneration: assessing associations with disease risk, severity of tau pathology, and clinical features Clusterin ameliorates tau pathology in vivo by inhibiting fibril formation Astrocyte-derived clusterin suppresses amyloid formation in vivo Apoptotic Neuron-Derived Histone Amyloid Fibrils Induce α-Synuclein Aggregation Associations of mitochondrial genomic variation with corticobasal degeneration, progressive supranuclear palsy, and neuropathological tau measures Orthostatic hypotension preceding dementia with Lewy bodies by over 15 years: a clinicopathologic case report Correction to: Deciphering cellular transcriptional alterations in Alzheimer’s disease brains Deciphering cellular transcriptional alterations in Alzheimer’s disease brains Correction to: Loss of homeostatic microglial phenotype in CSF1R-related Leukoencephalopathy Pick’s disease: clinicopathologic characterization of 21 cases Loss of homeostatic microglial phenotype in CSF1R-related Leukoencephalopathy TDP-43 is associated with a reduced likelihood of rendering a clinical diagnosis of dementia with Lewy bodies in autopsy-confirmed cases of transitional/diffuse Lewy body disease

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Dennis W. Dickson

Author Correction: Genetic, transcriptomic, histological, and biochemical analysis of progressive supranuclear palsy implicates glial activation and novel risk genes

Author Correction: Divergent single cell transcriptome and epigenome alterations in ALS and FTD patients with C9orf72 mutation

Genetic, transcriptomic, histological, and biochemical analysis of progressive supranuclear palsy implicates glial activation and novel risk genes

Correction: HDGFL2 cryptic proteins report presence of TDP-43 pathology in neurodegenerative diseases

Gliovascular transcriptional perturbations in Alzheimer’s disease reveal molecular mechanisms of blood brain barrier dysfunction

Abundant transcriptomic alterations in the human cerebellum of patients with a C9orf72 repeat expansion

Rare genetic variation in fibronectin 1 (FN1) protects against APOEε4 in Alzheimer’s disease

A multimodal clinical diagnostic approach using MRI and 18F-FDG-PET for antemortem diagnosis of TDP-43 in cases with low–intermediate Alzheimer’s disease neuropathologic changes and primary age-related tauopathy

Influences of amyloid-β and tau on white matter neurite alterations in dementia with Lewy bodies

A systematic review of progranulin concentrations in biofluids in over 7,000 people—assessing the pathogenicity of GRN mutations and other influencing factors

HDGFL2 cryptic proteins report presence of TDP-43 pathology in neurodegenerative diseases

Role of GBA variants in Lewy body disease neuropathology

Impact of APOE on amyloid and tau accumulation in argyrophilic grain disease and Alzheimer’s disease

Cryptic splicing of stathmin-2 and UNC13A mRNAs is a pathological hallmark of TDP-43-associated Alzheimer’s disease

Letter to the editor on: Hornerin deposits in neuronal intranuclear inclusion disease: direct identification of proteins with compositionally biased regions in inclusions by Park et al. (2022)

Cross species systems biology discovers glial DDR2, STOM, and KANK2 as therapeutic targets in progressive supranuclear palsy

Divergent single cell transcriptome and epigenome alterations in ALS and FTD patients with C9orf72 mutation

TDP-43-regulated cryptic RNAs accumulate in Alzheimer’s disease brains

Publisher Correction to: Diffuse argyrophilic grain disease with TDP-43 proteinopathy and neuronal intermediate filament inclusion disease: FTLD with mixed tau, TDP-43 and FUS pathologies

Diffuse argyrophilic grain disease with TDP-43 proteinopathy and neuronal intermediate filament inclusion disease: FTLD with mixed tau, TDP-43 and FUS pathologies

Case report of a patient with unclassified tauopathy with molecular and neuropathological features of both progressive supranuclear palsy and corticobasal degeneration

Cancer in pathologically confirmed multiple system atrophy

Genome-wide association study of brain biochemical phenotypes reveals distinct genetic architecture of Alzheimer’s disease related proteins

Molecular fragment characteristics and distribution of tangle associated TDP-43 (TATs) and other TDP-43 lesions in Alzheimer’s disease

Global neuropathologic severity of Alzheimer’s disease and locus coeruleus vulnerability influences plasma phosphorylated tau levels

LATE-NC staging in routine neuropathologic diagnosis: an update

Nuclear import receptors are recruited by FG-nucleoporins to rescue hallmarks of TDP-43 proteinopathy

Structure-based discovery of small molecules that disaggregate Alzheimer’s disease tissue derived tau fibrils in vitro

Discrepancy between distribution of alpha-synuclein oligomers and Lewy-related pathology in Parkinson’s disease

Rainwater Charitable Foundation criteria for the neuropathologic diagnosis of progressive supranuclear palsy

Evidence of cerebellar TDP-43 loss of function in FTLD-TDP

Neuropathologic scales of cerebrovascular disease associated with diffusion changes on MRI

Mitochondrial genomic variation in dementia with Lewy bodies: association with disease risk and neuropathological measures

Frequency of LATE neuropathologic change across the spectrum of Alzheimer’s disease neuropathology: combined data from 13 community-based or population-based autopsy cohorts

Frequency and distribution of TAR DNA-binding protein 43 (TDP-43) pathology increase linearly with age in a large cohort of older adults with and without dementia

Clinical and pathological characteristics of later onset multiple system atrophy

Proximity proteomics of C9orf72 dipeptide repeat proteins identifies molecular chaperones as modifiers of poly-GA aggregation

TDP-43 Pathology in Alzheimer’s Disease

Neuropathology and molecular diagnosis of Synucleinopathies

Correction to: Apolipoprotein E regulates lipid metabolism and α‑synuclein pathology in human iPSC‑derived cerebral organoids

Genome-wide association study and functional validation implicates JADE1 in tauopathy

Apolipoprotein E regulates lipid metabolism and α-synuclein pathology in human iPSC-derived cerebral organoids

Cellular and pathological heterogeneity of primary tauopathies

Predictors of cognitive impairment in primary age-related tauopathy: an autopsy study

Genome-wide analysis identifies a novel LINC-PINT splice variant associated with vascular amyloid pathology in Alzheimer’s disease

Tau isoforms are differentially expressed across the hippocampus in chronic traumatic encephalopathy and Alzheimer’s disease

Latent trait modeling of tau neuropathology in progressive supranuclear palsy

Frequency of spinocerebellar ataxia mutations in patients with multiple system atrophy

MAPT subhaplotypes in corticobasal degeneration: assessing associations with disease risk, severity of tau pathology, and clinical features

Clusterin ameliorates tau pathology in vivo by inhibiting fibril formation

Astrocyte-derived clusterin suppresses amyloid formation in vivo

Apoptotic Neuron-Derived Histone Amyloid Fibrils Induce α-Synuclein Aggregation

Associations of mitochondrial genomic variation with corticobasal degeneration, progressive supranuclear palsy, and neuropathological tau measures

Orthostatic hypotension preceding dementia with Lewy bodies by over 15 years: a clinicopathologic case report

Correction to: Deciphering cellular transcriptional alterations in Alzheimer’s disease brains

Deciphering cellular transcriptional alterations in Alzheimer’s disease brains

Correction to: Loss of homeostatic microglial phenotype in CSF1R-related Leukoencephalopathy

Pick’s disease: clinicopathologic characterization of 21 cases

Loss of homeostatic microglial phenotype in CSF1R-related Leukoencephalopathy

TDP-43 is associated with a reduced likelihood of rendering a clinical diagnosis of dementia with Lewy bodies in autopsy-confirmed cases of transitional/diffuse Lewy body disease