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Christine Skerka

Immunologin

Name	Christine Skerka
Ort	Jena (Wirkungsort) Deutschland
Geschlecht	weiblich
Akademischer Grad bzw. Titel	Prof. Dr. rer. nat.
Beruf	Immunologin (charakteristischer Beruf)
Variante	C. Skerka Christine Skerka-Zipfel (späterer Name) C. Skerka-Zipfel Christine S.- Zipfel
Familie	Peter F. Zipfel (Ehemann)
Beziehung	Leibniz-Institut für Naturstoff-Forschung und Infektionsbiologie
Weitere Angaben	Dissertation, Universität Bremen, 1989 Professur für Immunregulation an der Friedrich-Schiller-Universität Jena (FSU)
Datensatztyp	Person
GND-Nummer	1216431868
Datensatz-ID	1216431868
Beteiligt an alle anzeigen immune response of human monocytes to Lichtheimia corymbifera Analysis of human extracellular vesicles induced by Candida albicans specific role of neutrophil- and epithelial cell-derived extracellular vesicles in antifungal defence against Aspergillus fumigatus Modulation of human monocytes by the pathogenic fungus Candida albicans Borrelia valaisiana resist complement-mediated killing independently of the recruitment of immune regulators and inactivation of complement components Staphylococcus aureus proteins Sbi and Efb recruit human plasmin to degrade complement C3 and C3b Complement factor H-related proteins CFHR2 and CFHR5 represent novel ligands for the infection-associated CRASP proteins of Borrelia burgdorferi Immune evasion of Borrelia burgdorferi by acquisition of human complement regulators FHL‐1/reconectin and Factor H

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Christine Skerka

immune response of human monocytes to Lichtheimia corymbifera

Analysis of human extracellular vesicles induced by Candida albicans

specific role of neutrophil- and epithelial cell-derived extracellular vesicles in antifungal defence against Aspergillus fumigatus

Modulation of human monocytes by the pathogenic fungus Candida albicans

Borrelia valaisiana resist complement-mediated killing independently of the recruitment of immune regulators and inactivation of complement components

Staphylococcus aureus proteins Sbi and Efb recruit human plasmin to degrade complement C3 and C3b

Complement factor H-related proteins CFHR2 and CFHR5 represent novel ligands for the infection-associated CRASP proteins of Borrelia burgdorferi

Immune evasion of Borrelia burgdorferi by acquisition of human complement regulators FHL‐1/reconectin and Factor H