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Reinier Boon

Name	Reinier Boon
Ort	Frankfurt am Main (Wirkungsort) Deutschland
Geschlecht	männlich
Akademischer Grad bzw. Titel	Prof. Dr.
Variante	Reinier A. Boon
Beziehung	Goethe-Universität Frankfurt am Main Kardiologie (Thema)
Datensatztyp	Person
GND-Nummer	1222622718
Datensatz-ID	1222622718
Beteiligt an alle anzeigen Aging-regulated PNUTS maintains endothelial barrier function via SEMA3B suppression Changes in RNA dynamics in the course of hypoxia in myeloid cells Die tumorsuppressive MicroRNA-193b in Wirkstoff induzierter Differenzierung und Proliferation in Akuter Myeloischer und Promyeloischer Leukämie Endothelial microRNAs and long noncoding RNAs in cardiovascular ageing Impaired H19 lncRNA expression contributes to the compromised developmental angiogenesis in EVL-deficient mice Targeting long non-coding RNA NUDT6 enhances smooth muscle cell survival and limits vascular disease progression The circular RNA Ataxia Telangiectasia Mutated regulates oxidative stress in smooth muscle cells in expanding abdominal aortic aneurysms The splicing‐regulatory lncRNA NTRAS sustains vascular integrity Long noncoding RNAs in cardiometabolic disorders The endothelial-specific LINC00607 mediates endothelial angiogenic function The splicing-regulatory lncRNA NTRAS sustains vascular integrity Untersuchung von long non-coding RNA im Entzündungsmodell mit mesenchymalen Stamm-/Stromazellen Cardiomyocytes stimulate angiogenesis after ischemic injury in a ZEB2-dependent manner Cardiomyocytes stimulate angiogenesis after ischemic injury in a ZEB2-dependent manner LncRNA AERRIE is required for sulfatase 1 expression, but not for endothelial-to-mesenchymal transition Long non-coding RNA aerrie controls DNA damage repair via YBX1 to maintain endothelial cell function The Long Non-coding Road to Atherosclerosis The PNUTS-PP1 axis regulates endothelial aging and barrier function via SEMA3B suppression Long noncoding RNAs in key cellular processes involved in aortic aneurysms The lncRNA GATA6-AS epigenetically regulates endothelial gene expression via interaction with LOXL2 The lncRNA GATA6-AS epigenetically regulates endothelial gene expression via interaction with LOXL2

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Katalog der Deutschen Nationalbibliothek

Reinier Boon

Aging-regulated PNUTS maintains endothelial barrier function via SEMA3B suppression

Changes in RNA dynamics in the course of hypoxia in myeloid cells

Die tumorsuppressive MicroRNA-193b in Wirkstoff induzierter Differenzierung und Proliferation in Akuter Myeloischer und Promyeloischer Leukämie

Endothelial microRNAs and long noncoding RNAs in cardiovascular ageing

Impaired H19 lncRNA expression contributes to the compromised developmental angiogenesis in EVL-deficient mice

Targeting long non-coding RNA NUDT6 enhances smooth muscle cell survival and limits vascular disease progression

The circular RNA Ataxia Telangiectasia Mutated regulates oxidative stress in smooth muscle cells in expanding abdominal aortic aneurysms

The splicing‐regulatory lncRNA NTRAS sustains vascular integrity

Long noncoding RNAs in cardiometabolic disorders

The endothelial-specific LINC00607 mediates endothelial angiogenic function

The splicing-regulatory lncRNA NTRAS sustains vascular integrity

Untersuchung von long non-coding RNA im Entzündungsmodell mit mesenchymalen Stamm-/Stromazellen

Cardiomyocytes stimulate angiogenesis after ischemic injury in a ZEB2-dependent manner

Cardiomyocytes stimulate angiogenesis after ischemic injury in a ZEB2-dependent manner

LncRNA AERRIE is required for sulfatase 1 expression, but not for endothelial-to-mesenchymal transition

Long non-coding RNA aerrie controls DNA damage repair via YBX1 to maintain endothelial cell function

The Long Non-coding Road to Atherosclerosis

The PNUTS-PP1 axis regulates endothelial aging and barrier function via SEMA3B suppression

Long noncoding RNAs in key cellular processes involved in aortic aneurysms

The lncRNA GATA6-AS epigenetically regulates endothelial gene expression via interaction with LOXL2

The lncRNA GATA6-AS epigenetically regulates endothelial gene expression via interaction with LOXL2