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Irene Gonzalez Menendez

Pathologin

Name	Irene Gonzalez Menendez
Ort	Sonstiges Ausland / Unbekanntes Land
Geschlecht	weiblich
Akademischer Grad bzw. Titel	Dr.
Beruf	Pathologin (charakteristischer Beruf)
Variante	Irene Gonzalez-Menendez Irene Gonzalez Menendez
Weitere Angaben	Research Scientist bei Mouse Pathology University of Tübingen; Institute of Pathology and Neuropathology and Comprehensive Cancer Center, University of Tübingen; Postdoctoral Researcher, UKT - Universitätsklinikum Tübingen
Datensatztyp	Person
GND-Nummer	1222750619
Datensatz-ID	1222750619
Beteiligt an alle anzeigen Western diet increases brain metabolism and adaptive immune responses in a mouse model of amyloidosis Acidosis-mediated increase in IFN-γ-induced PD-L1 expression on cancer cells as an immune escape mechanism in solid tumors Acute and chronic inflammation alter immunometabolism in a cutaneous delayed-type hypersensitivity reaction (DTHR) mouse model Immunomodulatory role of reactive oxygen species and nitrogen species during T cell-driven neutrophil-enriched acute and chronic cutaneous delayed-type hypersensitivity reactions Existence of reprogrammed lymphoma stem cells in a murine ALCL-like model Existence of reprogrammed lymphoma stem cells in a murine ALCL-like model Temporal Dynamics of Reactive Oxygen and Nitrogen Species and NF-κB Activation During Acute and Chronic T Cell–Driven Inflammation CCR8 leads to eosinophil migration and regulates neutrophil migration in murine allergic enteritis Cysteine-type cathepsins promote the effector phase of acute cutaneous delayed-type hypersensitivity reactions Genetic deficiency of the tumor suppressor protein p53 influences erythrocyte survival

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Irene Gonzalez Menendez

Western diet increases brain metabolism and adaptive immune responses in a mouse model of amyloidosis

Acidosis-mediated increase in IFN-γ-induced PD-L1 expression on cancer cells as an immune escape mechanism in solid tumors

Acute and chronic inflammation alter immunometabolism in a cutaneous delayed-type hypersensitivity reaction (DTHR) mouse model

Immunomodulatory role of reactive oxygen species and nitrogen species during T cell-driven neutrophil-enriched acute and chronic cutaneous delayed-type hypersensitivity reactions

Existence of reprogrammed lymphoma stem cells in a murine ALCL-like model

Existence of reprogrammed lymphoma stem cells in a murine ALCL-like model

Temporal Dynamics of Reactive Oxygen and Nitrogen Species and NF-κB Activation During Acute and Chronic T Cell–Driven Inflammation

CCR8 leads to eosinophil migration and regulates neutrophil migration in murine allergic enteritis

Cysteine-type cathepsins promote the effector phase of acute cutaneous delayed-type hypersensitivity reactions

Genetic deficiency of the tumor suppressor protein p53 influences erythrocyte survival