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Bianca Dufner

Chemisch-technische Assistentin

Name	Bianca Dufner
Ort	Deutschland
Beruf	Chemisch-technische Assistentin (charakteristischer Beruf)
Weitere Angaben	Mitarb. Universitäts-Herzzentrum Bad Krozingen
Quelle	FreiDok plus
Datensatztyp	Person
GND-Nummer	1224613155
Datensatz-ID	1224613155
Beteiligt an alle anzeigen A comparative gene expression matrix in Apoe-deficient mice identifies unique and atherosclerotic disease stage-specific gene regulation patterns in monocytes and macrophages Elevated platelet–leukocyte complexes are associated with, but dispensable for myocardial ischemia–reperfusion injury Elevated platelet–leukocyte complexes are associated with, but dispensable for myocardial ischemia–reperfusion injury Myeloid cell-specific Irf5 deficiency stabilizes atherosclerotic plaques in Apoe–/– mice Inhibition of macrophage proliferation dominates plaque regression in response to cholesterol lowering Inhibition of macrophage proliferation dominates plaque regression in response to cholesterol lowering Atheroprotection through SYK inhibition fails in established disease when local macrophage proliferation dominates lesion progression Atheroprotection through SYK inhibition fails in established disease when local macrophage proliferation dominates lesion progression

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Bianca Dufner

A comparative gene expression matrix in Apoe-deficient mice identifies unique and atherosclerotic disease stage-specific gene regulation patterns in monocytes and macrophages

Elevated platelet–leukocyte complexes are associated with, but dispensable for myocardial ischemia–reperfusion injury

Elevated platelet–leukocyte complexes are associated with, but dispensable for myocardial ischemia–reperfusion injury

Myeloid cell-specific Irf5 deficiency stabilizes atherosclerotic plaques in Apoe–/– mice

Inhibition of macrophage proliferation dominates plaque regression in response to cholesterol lowering

Inhibition of macrophage proliferation dominates plaque regression in response to cholesterol lowering

Atheroprotection through SYK inhibition fails in established disease when local macrophage proliferation dominates lesion progression

Atheroprotection through SYK inhibition fails in established disease when local macrophage proliferation dominates lesion progression