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Angela Brieger (1967-)

Name	Angela Brieger
Zeit	1967- (Lebensdaten)
Akademischer Grad bzw. Titel	Dr. phil. nat. (am Fachbereich Biochemie, Pharmazie und Lebensmittelchemie der Universität Frankfurt (Main)) (2000)
Weitere Angaben	Habil.-Schr. am Fachbereich Medizin (Fach Molekulare Medizin) der Universität Frankfurt (Main), 2012
Datensatztyp	Person
GND-Nummer	122829697
Datensatz-ID	122829697
Beteiligt an alle anzeigen Erk inhibition as a promising therapeutic strategy for high IL-8-secreting and low SPTAN1-expressing colorectal cancer Key role of phosphorylation sites in ATPase domain and Linker region of MLH1 for DNA binding and functionality of MutLα Key role of phosphorylation sites in ATPase domain and Linker region of MLH1 for DNA binding and functionality of MutLα High expression of casein kinase 2 alpha is responsible for enhanced phosphorylation of DNA mismatch repair protein MLH1 and increased tumor mutation rates in colorectal cancer Investigation of the Wilson gene ATP7B transcriptional start site and the effect of core promoter alterations SPTAN1 expression predicts treatment and survival outcomes in colorectal cancer Alternative AKT2 splicing produces protein lacking the hydrophobic motif regulatory region Downregulation of SPTAN1 is related to MLH1 deficiency and metastasis in colorectal cancer The role of nonerythroid spectrin αII in cancer Exome sequencing identifies biallelic MSH3 germline mutations as a recessive subtype of colorectal adenomatous polyposis Reduced migration of MLH1 deficient colon cancer cells depends on SPTAN1 Promoter methylation of MLH1, PMS2, MSH2 and p16 is a phenomenon of advanced-stage HCCs Reduced migration of MLH1 deficient colon cancer cells depends on SPTAN1 C-terminal fluorescent labeling impairs functionality of DNA mismatch repair proteins Intrazellulärer Transport des humanen DNA-Mismatch-Reparaturproteins MLH1 und Untersuchung seiner Interaktion mit Reparatur-unabhängigen Proteinen Cytoskeletal scaffolding proteins interact with Lynch‐Syndrome associated mismatch repair protein MLH1 Cytoskeletal scaffolding proteins interact with Lynch‐Syndrome associated mismatch repair protein MLH1 N‐terminus of hMLH1 confers interaction of hMutLα and hMutLβ with hMutSα BAX and caspase-5 frameshift mutations and spontaneous apoptosis in colorectal cancer with microsatellite instability Nachweis und funktionelle Charakterisierung der mismatch repair Gene, hMLH1 und hMSH2, beim erblichen nicht polypösen kolorektalen Karzinom

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Angela Brieger (1967-)

Erk inhibition as a promising therapeutic strategy for high IL-8-secreting and low SPTAN1-expressing colorectal cancer

Key role of phosphorylation sites in ATPase domain and Linker region of MLH1 for DNA binding and functionality of MutLα

Key role of phosphorylation sites in ATPase domain and Linker region of MLH1 for DNA binding and functionality of MutLα

High expression of casein kinase 2 alpha is responsible for enhanced phosphorylation of DNA mismatch repair protein MLH1 and increased tumor mutation rates in colorectal cancer

Investigation of the Wilson gene ATP7B transcriptional start site and the effect of core promoter alterations

SPTAN1 expression predicts treatment and survival outcomes in colorectal cancer

Alternative AKT2 splicing produces protein lacking the hydrophobic motif regulatory region

Downregulation of SPTAN1 is related to MLH1 deficiency and metastasis in colorectal cancer

The role of nonerythroid spectrin αII in cancer

Exome sequencing identifies biallelic MSH3 germline mutations as a recessive subtype of colorectal adenomatous polyposis

Reduced migration of MLH1 deficient colon cancer cells depends on SPTAN1

Promoter methylation of MLH1, PMS2, MSH2 and p16 is a phenomenon of advanced-stage HCCs

Reduced migration of MLH1 deficient colon cancer cells depends on SPTAN1

C-terminal fluorescent labeling impairs functionality of DNA mismatch repair proteins

Intrazellulärer Transport des humanen DNA-Mismatch-Reparaturproteins MLH1 und Untersuchung seiner Interaktion mit Reparatur-unabhängigen Proteinen

Cytoskeletal scaffolding proteins interact with Lynch‐Syndrome associated mismatch repair protein MLH1

Cytoskeletal scaffolding proteins interact with Lynch‐Syndrome associated mismatch repair protein MLH1

N‐terminus of hMLH1 confers interaction of hMutLα and hMutLβ with hMutSα

BAX and caspase-5 frameshift mutations and spontaneous apoptosis in colorectal cancer with microsatellite instability

Nachweis und funktionelle Charakterisierung der mismatch repair Gene, hMLH1 und hMSH2, beim erblichen nicht polypösen kolorektalen Karzinom