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Cemil Kerimoglu

Neurowissenschaftler

Name	Cemil Kerimoglu
Zeit	2012- (Wirkungsdaten)
Ort	Bonn (Wirkungsort) Deutschland
Geschlecht	männlich
Akademischer Grad bzw. Titel	Dr. (Georg-August-Universität Göttingen) (2012)
Beruf	Neurowissenschaftler (charakteristischer Beruf)
Variante	C. Kerimoglu Cemil Kerimolgu Kerimoglu Cemil
Beziehung	Deutsches Zentrum für Neurodegenerative Erkrankungen
Weitere Angaben	Institute for Neuroanatomy, University Medical Center Goettingen; Department of Human Genetics, Ruhr University of Bochum; German Center for Neurodegenerative Diseases, Göttingen
Quelle	ResearchGate: https://www.researchgate.net/profile/Kerimoglu-Cemil (Stand: 28.07.2023) http://www.fischerlab.uni-goettingen.de/lab-members.php (Stand: 28.07.2023)
Datensatztyp	Person
GND-Nummer	1245481487
Datensatz-ID	1245481487
Beteiligt an alle anzeigen Postnatal expression of the lysine methyltransferase SETD1B is essential for learning and the regulation of neuron‐enriched genes A microRNA signature that correlates with cognition and is a target against cognitive decline Conditional loss of BAF (mSWI/SNF) scaffolding subunits affects specification and proliferation of oligodendrocyte precursors in developing mouse forebrain Exercise as a model to identify microRNAs linked to human cognition: a role for microRNA-409 and microRNA-501 Loss of BAF complex in developing cortex perturbs radial neuronal migration in a WNT signaling-dependent manner Innate immune memory in the brain shapes neurological disease hallmarks Formin 2 links neuropsychiatric phenotypes at young age to an increased risk for dementia K‐Lysine acetyltransferase 2a regulates a hippocampal gene expression network linked to memory formation Role of Histone Methylation in Cognition and Effects of Different Durations of Environmental Enrichment on Learning and Memory

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Cemil Kerimoglu

Postnatal expression of the lysine methyltransferase SETD1B is essential for learning and the regulation of neuron‐enriched genes

A microRNA signature that correlates with cognition and is a target against cognitive decline

Conditional loss of BAF (mSWI/SNF) scaffolding subunits affects specification and proliferation of oligodendrocyte precursors in developing mouse forebrain

Exercise as a model to identify microRNAs linked to human cognition: a role for microRNA-409 and microRNA-501

Loss of BAF complex in developing cortex perturbs radial neuronal migration in a WNT signaling-dependent manner

Innate immune memory in the brain shapes neurological disease hallmarks

Formin 2 links neuropsychiatric phenotypes at young age to an increased risk for dementia

K‐Lysine acetyltransferase 2a regulates a hippocampal gene expression network linked to memory formation

Role of Histone Methylation in Cognition and Effects of Different Durations of Environmental Enrichment on Learning and Memory