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Angelika Lampert (1975-)

Physiologin

Name	Angelika Lampert
Zeit	1975- (Lebensdaten)
Ort	Nürnberg (Geburtsort) Aachen (Wirkungsort) Deutschland
Geschlecht	weiblich
Akademischer Grad bzw. Titel	Prof. Dr. med.
Beruf	Physiologin (charakteristischer Beruf)
Variante	A. Lampert
Beziehung	Universitätsklinikum Aachen Friedrich-Schiller-Universität Jena. Medizinische Fakultät
Weitere Angaben	Dissertation, Friedrich-Schiller-Universität Jena, 2003
Datensatztyp	Person
GND-Nummer	124639747
Datensatz-ID	124639747
Beteiligt an alle anzeigen Increased anger and stress and heightened connectivity between IFG and vmPFC in victims during social interaction How to differentiate induced pluripotent stem cells into sensory neurons for disease modelling: a functional assessment The pain target NaV1.7 is expressed late during human iPS cell differentiation into sensory neurons as determined in high-resolution imaging Analgesic effect of Botulinum toxin in neuropathic pain is sodium channel independent Effekte von Ranolazin und Aprindin auf die Einzelkanalaktivität des spannungsabhängigen Natriumkanals Nav1.5 und dessen LQT3/CCDMutante T1620K Increased anger and stress and heightened connectivity between IFG and vmPFC in victims during social interaction Painful stimulation increases functional connectivity between supplementary motor area and thalamus in patients with small fibre neuropathy Reduced Gray Matter Volume and Cortical Thickness in Patients With Small-Fiber Neuropathy The pain target NaV1.7 is expressed late during human iPS cell differentiation into sensory neurons as determined in high-resolution imaging Toward the Next Generation of Neural Iontronic Interfaces Transformative Materials to Create 3D Functional Human Tissue Models In Vitro in a Reproducible Manner Neural correlates of multisensory integration in the human brain: an ALE meta-analysis Beyond mobile phone displays : Flat panel display technology for biomedical applications Cold and warmth intensify pain-linked sodium channel gating effects and persistent currents Corrigendum: ClC-3 regulates the excitability of nociceptive neurons and is involved in inflammatory processes within the spinal sensory pathway Corrigendum: Diclofenac and other non-steroidal anti-inflammatory drugs (NSAIDs) are competitive antagonists of the human P2X3 receptor Development of a 3D innervated skin model integrating anisotrophic guidance elements Diclofenac and other non-steroidal anti-inflammatory drugs (NSAIDs) are competitive antagonists of the human P2X3 receptor Updated Efficient neuronal processing mechanisms in the olfactory system & insights into impaired brain function in autism spectrum disorder using SHANK3 mice Genetic landscape of congenital insensitivity to pain and hereditary sensory and autonomic neuropathies Toward the Next Generation of Neural Iontronic Interfaces Transformative Materials to Create 3D Functional Human Tissue Models In Vitro in a Reproducible Manner Nociception, Transcriptomics ET CETERA: NOCICEPTRA Functional subgroups of rat and human sensory neurons: a systematic review of electrophysiological properties ClC-3 regulates the excitability of nociceptive neurons and is involved in inflammatory processes within the spinal sensory pathway Experimental und environmental factors influencing human perception of direct current-, alternating current-, and hybrid electric fields Functional subgroups of rat and human sensory neurons: a systematic review of electrophysiological properties Identification of essential gene products required for the maintenance of adult neurons in Drosophila melanogaster Investigating the function of individual subunits in trimeric P2X7 receptors Nociception, Transcriptomics ET CETERA: NOCICEPTRA Pharmacological modification of sodium channel Nav1.9 activity Sodium Channels and Local Anesthetics—Old Friends With New Perspectives Sodium ion channels in chronic pain: modeling human pain syndromes with induced pluripotent stem cell-derived sensory neurons The spatial self-organization within pluripotent stem cell colonies is continued in detaching aggregates Biocompatible Micron‐Scale Silk Fibers Fabricated by Microfluidic Wet Spinning Assessing the impact of pain-linked Nav1.7 variants : An example of two variants with no biophysical effect Biocompatible Micron-Scale Silk Fibers Fabricated by Microfluidic Wet Spinning Biocompatible Micron‐Scale Silk Fibers Fabricated by Microfluidic Wet Spinning Grenzen der Messbarkeit bei der Analyse des Schaltens von HCN2-Kanälen The difficulty to model Huntington’s disease in vitro using striatal medium spiny neurons differentiated from human induced pluripotent stem cells The Expression of the Cancer-Associated lncRNA Snhg15 Is Modulated by EphrinA5-Induced Signaling The insecticide deltamethrin enhances sodium channel slow inactivation of human Nav1.9, Nav1.8 and Nav1.7 The Potential Effect of Nav1.8 in Autism Spectrum Disorder : Evidence From a Congenital Case With Compound Heterozygous SCN10A Mutations The role of voltage-gated sodium channels in neurological diseases : lessons learned from pain disorders and Huntington’s Disease NGF-Induced Nav1.7 Upregulation Contributes to Chronic Post-surgical Pain by Activating SGK1-Dependent Nedd4-2 Phosphorylation PRDM8 reveals aberrant DNA methylation in aging syndromes and is relevant for hematopoietic and neuronal differentiation Effect of odor pleasantness on heat-induced pain: An fMRI study Acquisition of chromosome 1q duplication in parental and genome-edited human-induced pluripotent stem cell-derived neural stem cells results in their higher proliferation rate in vitro and in vivo Characterization of Synthetic Tf2 as a Na$_{V}$1.3 Selective Pharmacological Probe Dataset of electrophysiological patch-clamp recordings of the effect of the compounds deltamethrin, ATx-II and β4-peptide on human cardiac Nav1.5 sodium channel gating properties Methylglyoxal verursacht Schmerz und Hyperalgesie durch Aktivierung humaner C-Fasern Phosphorylation of a chronic pain mutation in the voltage-gated sodium channel Nav1.7 increases voltage sensitivity Uncoupling sodium channel dimers restores the phenotype of a pain‐linked Nav1.7 channel mutation β1 and β3 subunits amplify mechanosensitivity of the cardiac voltage-gated sodium channel Nav1.5 Noncanonical Ion Channel Behaviour in Pain Noncanonical Ion Channel Behaviour in Pain Pain relief in a neuropathy patient by lacosamide: Proof of principle of clinical translation from patient-specific iPS cell-derived nociceptors The role of Nav1.7 in human nociceptors: insights from human induced pluripotent stem cell–derived sensory neurons of erythromelalgia patients Neuropathische Schmerzsyndrome bei Ionenkanalerkrankungen Loss-of-function of Nav1.8/D1639N linked to human pain can be rescued by lidocaine

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Katalog der Deutschen Nationalbibliothek

Angelika Lampert (1975-)

Increased anger and stress and heightened connectivity between IFG and vmPFC in victims during social interaction

How to differentiate induced pluripotent stem cells into sensory neurons for disease modelling: a functional assessment

The pain target NaV1.7 is expressed late during human iPS cell differentiation into sensory neurons as determined in high-resolution imaging

Analgesic effect of Botulinum toxin in neuropathic pain is sodium channel independent

Effekte von Ranolazin und Aprindin auf die Einzelkanalaktivität des spannungsabhängigen Natriumkanals Nav1.5 und dessen LQT3/CCDMutante T1620K

Increased anger and stress and heightened connectivity between IFG and vmPFC in victims during social interaction

Painful stimulation increases functional connectivity between supplementary motor area and thalamus in patients with small fibre neuropathy

Reduced Gray Matter Volume and Cortical Thickness in Patients With Small-Fiber Neuropathy

The pain target NaV1.7 is expressed late during human iPS cell differentiation into sensory neurons as determined in high-resolution imaging

Toward the Next Generation of Neural Iontronic Interfaces

Transformative Materials to Create 3D Functional Human Tissue Models In Vitro in a Reproducible Manner

Neural correlates of multisensory integration in the human brain: an ALE meta-analysis

Beyond mobile phone displays : Flat panel display technology for biomedical applications

Cold and warmth intensify pain-linked sodium channel gating effects and persistent currents

Corrigendum: ClC-3 regulates the excitability of nociceptive neurons and is involved in inflammatory processes within the spinal sensory pathway

Corrigendum: Diclofenac and other non-steroidal anti-inflammatory drugs (NSAIDs) are competitive antagonists of the human P2X3 receptor

Development of a 3D innervated skin model integrating anisotrophic guidance elements

Diclofenac and other non-steroidal anti-inflammatory drugs (NSAIDs) are competitive antagonists of the human P2X3 receptor Updated

Efficient neuronal processing mechanisms in the olfactory system & insights into impaired brain function in autism spectrum disorder using SHANK3 mice

Genetic landscape of congenital insensitivity to pain and hereditary sensory and autonomic neuropathies

Toward the Next Generation of Neural Iontronic Interfaces

Transformative Materials to Create 3D Functional Human Tissue Models In Vitro in a Reproducible Manner

Nociception, Transcriptomics ET CETERA: NOCICEPTRA

Functional subgroups of rat and human sensory neurons: a systematic review of electrophysiological properties

ClC-3 regulates the excitability of nociceptive neurons and is involved in inflammatory processes within the spinal sensory pathway

Experimental und environmental factors influencing human perception of direct current-, alternating current-, and hybrid electric fields

Functional subgroups of rat and human sensory neurons: a systematic review of electrophysiological properties

Identification of essential gene products required for the maintenance of adult neurons in Drosophila melanogaster

Investigating the function of individual subunits in trimeric P2X7 receptors

Nociception, Transcriptomics ET CETERA: NOCICEPTRA

Pharmacological modification of sodium channel Nav1.9 activity

Sodium Channels and Local Anesthetics—Old Friends With New Perspectives

Sodium ion channels in chronic pain: modeling human pain syndromes with induced pluripotent stem cell-derived sensory neurons

The spatial self-organization within pluripotent stem cell colonies is continued in detaching aggregates

Biocompatible Micron‐Scale Silk Fibers Fabricated by Microfluidic Wet Spinning

Assessing the impact of pain-linked Nav1.7 variants : An example of two variants with no biophysical effect

Biocompatible Micron-Scale Silk Fibers Fabricated by Microfluidic Wet Spinning

Biocompatible Micron‐Scale Silk Fibers Fabricated by Microfluidic Wet Spinning

Grenzen der Messbarkeit bei der Analyse des Schaltens von HCN2-Kanälen

The difficulty to model Huntington’s disease in vitro using striatal medium spiny neurons differentiated from human induced pluripotent stem cells

The Expression of the Cancer-Associated lncRNA Snhg15 Is Modulated by EphrinA5-Induced Signaling

The insecticide deltamethrin enhances sodium channel slow inactivation of human Nav1.9, Nav1.8 and Nav1.7

The Potential Effect of Nav1.8 in Autism Spectrum Disorder : Evidence From a Congenital Case With Compound Heterozygous SCN10A Mutations

The role of voltage-gated sodium channels in neurological diseases : lessons learned from pain disorders and Huntington’s Disease

NGF-Induced Nav1.7 Upregulation Contributes to Chronic Post-surgical Pain by Activating SGK1-Dependent Nedd4-2 Phosphorylation

PRDM8 reveals aberrant DNA methylation in aging syndromes and is relevant for hematopoietic and neuronal differentiation

Effect of odor pleasantness on heat-induced pain: An fMRI study

Acquisition of chromosome 1q duplication in parental and genome-edited human-induced pluripotent stem cell-derived neural stem cells results in their higher proliferation rate in vitro and in vivo

Characterization of Synthetic Tf2 as a Na$_{V}$1.3 Selective Pharmacological Probe

Dataset of electrophysiological patch-clamp recordings of the effect of the compounds deltamethrin, ATx-II and β4-peptide on human cardiac Nav1.5 sodium channel gating properties

Methylglyoxal verursacht Schmerz und Hyperalgesie durch Aktivierung humaner C-Fasern

Phosphorylation of a chronic pain mutation in the voltage-gated sodium channel Nav1.7 increases voltage sensitivity

Uncoupling sodium channel dimers restores the phenotype of a pain‐linked Nav1.7 channel mutation

β1 and β3 subunits amplify mechanosensitivity of the cardiac voltage-gated sodium channel Nav1.5

Noncanonical Ion Channel Behaviour in Pain

Noncanonical Ion Channel Behaviour in Pain

Pain relief in a neuropathy patient by lacosamide: Proof of principle of clinical translation from patient-specific iPS cell-derived nociceptors

The role of Nav1.7 in human nociceptors: insights from human induced pluripotent stem cell–derived sensory neurons of erythromelalgia patients

Neuropathische Schmerzsyndrome bei Ionenkanalerkrankungen

Loss-of-function of Nav1.8/D1639N linked to human pain can be rescued by lidocaine