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Rune Hartmann

Molekularbiologe

Name	Rune Hartmann
Zeit	1997- (Wirkungsdaten)
Ort	Dänemark
Geschlecht	männlich
Akademischer Grad bzw. Titel	Prof. Dr.
Beruf	Molekularbiologe (charakteristischer Beruf) Hochschullehrer
Beziehung	Aarhus Universitet
Quelle	https://pure.au.dk/portal/en/persons/rh%40mbg.au.dk (Stand: 25.11.2024)
Datensatztyp	Person
GND-Nummer	126030857X
Datensatz-ID	126030857X
Beteiligt an alle anzeigen Viral recognition and the antiviral interferon response Cryo‐EM structure of the human NKCC1 transporter reveals mechanisms of ion coupling and specificity Interferon-λ improves the efficacy of intranasally or rectally administered influenza subunit vaccines by a thymic stromal lymphopoietin-dependent mechanism Author Correction: STEEP mediates STING ER exit and activation of signaling STEEP mediates STING ER exit and activation of signaling SARS‐CoV‐2 evades immune detection in alveolar macrophages Defective interferon priming and impaired antiviral responses in a patient with an IRF7 variant and severe influenza Interferon-λ enhances adaptive mucosal immunity by boosting release of thymic stromal lymphopoietin Identification of an IRF3 variant and defective antiviral interferon responses in a patient with severe influenza IFN-[lambda] prevents influenza virus spread from the upper airways to the lungs and limits virus transmission Frequently used bioinformatics tools overestimate the damaging effect of allelic variants cGAS is activated by DNA in a length‐dependent manner HSV ‐1 ICP 27 targets the TBK 1‐activated STING signalsome to inhibit virus‐induced type I IFN expression IFN λ is a potent anti‐influenza therapeutic without the inflammatory side effects of IFN α treatment

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Rune Hartmann

Viral recognition and the antiviral interferon response

Cryo‐EM structure of the human NKCC1 transporter reveals mechanisms of ion coupling and specificity

Interferon-λ improves the efficacy of intranasally or rectally administered influenza subunit vaccines by a thymic stromal lymphopoietin-dependent mechanism

Author Correction: STEEP mediates STING ER exit and activation of signaling

STEEP mediates STING ER exit and activation of signaling

SARS‐CoV‐2 evades immune detection in alveolar macrophages

Defective interferon priming and impaired antiviral responses in a patient with an IRF7 variant and severe influenza

Interferon-λ enhances adaptive mucosal immunity by boosting release of thymic stromal lymphopoietin

Identification of an IRF3 variant and defective antiviral interferon responses in a patient with severe influenza

IFN-[lambda] prevents influenza virus spread from the upper airways to the lungs and limits virus transmission

Frequently used bioinformatics tools overestimate the damaging effect of allelic variants

cGAS is activated by DNA in a length‐dependent manner

HSV ‐1 ICP 27 targets the TBK 1‐activated STING signalsome to inhibit virus‐induced type I IFN expression

IFN λ is a potent anti‐influenza therapeutic without the inflammatory side effects of IFN α treatment