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Antonio F. Pardiñas

Dozent

Name	Antonio F. Pardiñas
Zeit	2022- (Wirkungsdaten)
Ort	Großbritannien
Geschlecht	männlich
Akademischer Grad bzw. Titel	Dr.
Beruf	Dozent (charakteristischer Beruf)
Variante	Antonio Pardiñas Antonio Fernández Pardiñas A. F. Pardiñas A. Pardiñas
Beziehung	Cardiff University Neurowissenschaften (Thema)
Weitere Angaben	Dozent der Cardiff University
Quelle	https://profiles.cardiff.ac.uk/staff/pardinasa (Stand: 30.04.2024)
Datensatztyp	Person
GND-Nummer	1327938987
Datensatz-ID	1327938987
Beteiligt an alle anzeigen Assessing the validity of a self-reported clinical diagnosis of schizophrenia Fine-mapping across diverse ancestries drives the discovery of putative causal variants underlying human complex traits and diseases Dopamine signaling enriched striatal gene set predicts striatal dopamine synthesis and physiological activity in vivo Common risk alleles for schizophrenia within the major histocompatibility complex predict white matter microstructure Mediation and longitudinal analysis to interpret the association between clozapine pharmacokinetics, pharmacogenomics, and absolute neutrophil count Gene set enrichment analysis of pathophysiological pathways highlights oxidative stress in psychosis A polygenic score indexing a DRD2-related co-expression network is associated with striatal dopamine function Genetic association of FMRP targets with psychiatric disorders Publisher Correction: Common schizophrenia alleles are enriched in mutation-intolerant genes and in regions under strong background selection Gene expression imputation across multiple brain regions provides insights into schizophrenia risk A genome-wide association study in individuals of African ancestry reveals the importance of the Duffy-null genotype in the assessment of clozapine-related neutropenia Expression quantitative trait loci in the developing human brain and their enrichment in neuropsychiatric disorders Genetic identification of brain cell types underlying schizophrenia Common schizophrenia alleles are enriched in mutation-intolerant genes and in regions under strong background selection

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Katalog der Deutschen Nationalbibliothek

Antonio F. Pardiñas

Assessing the validity of a self-reported clinical diagnosis of schizophrenia

Fine-mapping across diverse ancestries drives the discovery of putative causal variants underlying human complex traits and diseases

Dopamine signaling enriched striatal gene set predicts striatal dopamine synthesis and physiological activity in vivo

Common risk alleles for schizophrenia within the major histocompatibility complex predict white matter microstructure

Mediation and longitudinal analysis to interpret the association between clozapine pharmacokinetics, pharmacogenomics, and absolute neutrophil count

Gene set enrichment analysis of pathophysiological pathways highlights oxidative stress in psychosis

A polygenic score indexing a DRD2-related co-expression network is associated with striatal dopamine function

Genetic association of FMRP targets with psychiatric disorders

Publisher Correction: Common schizophrenia alleles are enriched in mutation-intolerant genes and in regions under strong background selection

Gene expression imputation across multiple brain regions provides insights into schizophrenia risk

A genome-wide association study in individuals of African ancestry reveals the importance of the Duffy-null genotype in the assessment of clozapine-related neutropenia

Expression quantitative trait loci in the developing human brain and their enrichment in neuropsychiatric disorders

Genetic identification of brain cell types underlying schizophrenia

Common schizophrenia alleles are enriched in mutation-intolerant genes and in regions under strong background selection