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Nils Schallner (1980-)

Arzt

Name	Nils Schallner
Zeit	1980- (Lebensdaten)
Ort	Köln (Geburtsort) Deutschland
Beruf	Arzt (charakteristischer Beruf) Postdoc
Beziehung	Klinik für Anästhesiologie und Intensivmedizin (Freiburg im Breisgau)
Weitere Angaben	Diss. 2006, Universität Freiburg, Medizinische Fakultät
Datensatztyp	Person
GND-Nummer	134256212
Datensatz-ID	134256212
Beteiligt an alle anzeigen Neuroprotection via carbon monoxide depends on the circadian regulation of CD36-mediated microglial erythrophagocytosis in hemorrhagic stroke The molecular mechanisms underlying the neuroprotective properties of heme oxygenase 1 and their connection to circadian rhythm after repetitive mild traumatic brain injury Verteilung und Transkriptionsprofil retinaler Mikrogliazellen im Mausmodell der sauerstoffinduzierten Retinopathie Circadian dependency of microglial heme oxygenase-1 expression and inflammation determine neuronal injury in hemorrhagic stroke Predictive markers related to local and systemic inflammation in severe COVID-19-associated ARDS: a prospective single-center analysis Circadian dependency of microglial heme oxygenase-1 expression and inflammation determine neuronal injury in hemorrhagic stroke Inzidenz- und Überlebensanalyse der zerebellären Blutung: Eine retrospektive Kohortenstudie Predictive markers related to local and systemic inflammation in severe COVID-19-associated ARDS: a prospective single-center analysis The role of heme and heme-oxygenase-1 in repetitive mild traumatic brain injury Usage of augmented reality for interventional neuraxial procedures : : a phantom-based study Early and Rapid Identification of COVID-19 Patients with Neutralizing Type I Interferon Auto-antibodies Bedeutung von Hämoxygenase-1 und CD36 bei der Erythrozyten-Phagozytose in Mikroglia Early and rapid identification of COVID-19 patients with neutralizing type I interferon auto-antibodies Intensive care unit resources and patient‐centred outcomes in severe COVID‐19: a prospective single‐centre economic evaluation T-cell dysfunction in the glioblastoma microenvironment is mediated by myeloid cells releasing interleukin-10 T-cell dysfunction in the glioblastoma microenvironment is mediated by myeloid cells releasing interleukin-10 Temporal expression pattern of hemoxygenase-1 expression and its association with vasospasm and delayed cerebral ischemia after aneurysmal subarachnoid hemorrhage Temporal Expression Pattern of Hemoxygenase-1 Expression and Its Association with Vasospasm and Delayed Cerebral Ischemia After Aneurysmal Subarachnoid Hemorrhage Expression of HO1 and PER2 can predict the incidence of delirium in trauma patients with concomitant brain injury Patients with subarachnoid hemorrhage exhibit disturbed expression patterns of the circadian rhythm gene period-2 Carbon monoxide controls microglial erythrophagocytosis by regulating CD36 surface expression to reduce the severity of hemorrhagic injury Neuroprotection after hemorrhagic stroke depends on cerebral heme oxygenase-1 The Carbon monoxide releasing molecule ALF-186 mediates anti-inflammatory and neuroprotective effects via the soluble guanylate cyclase ß1 in rats’ retinal ganglion cells after ischemia and reperfusion injury Argon inhalation attenuates retinal apoptosis after ischemia/reperfusion injury in a time- and dose-dependent manner in rats Argon inhalation attenuates retinal apoptosis after ischemia/reperfusion injury in a time- and dose-dependent manner in rats Carbon monoxide abrogates ischemic insult to neuronal cells via the soluble guanylate cyclase-cGMP pathway Postconditioning with inhaled carbon monoxide counteracts apoptosis and neuroinflammation in the ischemic rat retina Evaluation des diagnostischen und therapeutischen Potentials des aktivationsspezifischen, gegen das Leukozytenintegrin-Mac-1 (CD11b-CD18, αMβ2 [alpha M beta 2]) gerichteten, monoklonalen Einzelketten-Antikörper MAN-1 : funktionelle Charakterisierung und Ex-vivo-Evaluation an Sepsis-Patienten

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Katalog der Deutschen Nationalbibliothek

Nils Schallner (1980-)

Neuroprotection via carbon monoxide depends on the circadian regulation of CD36-mediated microglial erythrophagocytosis in hemorrhagic stroke

The molecular mechanisms underlying the neuroprotective properties of heme oxygenase 1 and their connection to circadian rhythm after repetitive mild traumatic brain injury

Verteilung und Transkriptionsprofil retinaler Mikrogliazellen im Mausmodell der sauerstoffinduzierten Retinopathie

Circadian dependency of microglial heme oxygenase-1 expression and inflammation determine neuronal injury in hemorrhagic stroke

Predictive markers related to local and systemic inflammation in severe COVID-19-associated ARDS: a prospective single-center analysis

Circadian dependency of microglial heme oxygenase-1 expression and inflammation determine neuronal injury in hemorrhagic stroke

Inzidenz- und Überlebensanalyse der zerebellären Blutung: Eine retrospektive Kohortenstudie

Predictive markers related to local and systemic inflammation in severe COVID-19-associated ARDS: a prospective single-center analysis

The role of heme and heme-oxygenase-1 in repetitive mild traumatic brain injury

Usage of augmented reality for interventional neuraxial procedures : : a phantom-based study

Early and Rapid Identification of COVID-19 Patients with Neutralizing Type I Interferon Auto-antibodies

Bedeutung von Hämoxygenase-1 und CD36 bei der Erythrozyten-Phagozytose in Mikroglia

Early and rapid identification of COVID-19 patients with neutralizing type I interferon auto-antibodies

Intensive care unit resources and patient‐centred outcomes in severe COVID‐19: a prospective single‐centre economic evaluation

T-cell dysfunction in the glioblastoma microenvironment is mediated by myeloid cells releasing interleukin-10

T-cell dysfunction in the glioblastoma microenvironment is mediated by myeloid cells releasing interleukin-10

Temporal expression pattern of hemoxygenase-1 expression and its association with vasospasm and delayed cerebral ischemia after aneurysmal subarachnoid hemorrhage

Temporal Expression Pattern of Hemoxygenase-1 Expression and Its Association with Vasospasm and Delayed Cerebral Ischemia After Aneurysmal Subarachnoid Hemorrhage

Expression of HO1 and PER2 can predict the incidence of delirium in trauma patients with concomitant brain injury

Patients with subarachnoid hemorrhage exhibit disturbed expression patterns of the circadian rhythm gene period-2

Carbon monoxide controls microglial erythrophagocytosis by regulating CD36 surface expression to reduce the severity of hemorrhagic injury

Neuroprotection after hemorrhagic stroke depends on cerebral heme oxygenase-1

The Carbon monoxide releasing molecule ALF-186 mediates anti-inflammatory and neuroprotective effects via the soluble guanylate cyclase ß1 in rats’ retinal ganglion cells after ischemia and reperfusion injury

Argon inhalation attenuates retinal apoptosis after ischemia/reperfusion injury in a time- and dose-dependent manner in rats

Argon inhalation attenuates retinal apoptosis after ischemia/reperfusion injury in a time- and dose-dependent manner in rats

Carbon monoxide abrogates ischemic insult to neuronal cells via the soluble guanylate cyclase-cGMP pathway

Postconditioning with inhaled carbon monoxide counteracts apoptosis and neuroinflammation in the ischemic rat retina

Evaluation des diagnostischen und therapeutischen Potentials des aktivationsspezifischen, gegen das Leukozytenintegrin-Mac-1 (CD11b-CD18, αMβ2 [alpha M beta 2]) gerichteten, monoklonalen Einzelketten-Antikörper MAN-1 : funktionelle Charakterisierung und Ex-vivo-Evaluation an Sepsis-Patienten