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Person

Aet O'Leary (1974-)

Psychologe

Name	Aet O'Leary
Zeit	1974- (Lebensdaten)
Ort	Estland
Beruf	Psychologe (charakteristischer Beruf)
Variante	Aet Alttoa (früherer Name)
Weitere Angaben	Tartu, Univ., Diss., 2008
Quelle	Vorlage
Datensatztyp	Person
GND-Nummer	139585265
Datensatz-ID	139585265
Beteiligt an alle anzeigen A systematic review of preclinical studies exploring the role of insulin signalling in executive function and memory A systematic review of preclinical studies exploring the role of insulin signalling in executive function and memory Insulin and disorders of behavioural flexibility Behavioural and functional evidence revealing the role of RBFOX1 variation in multiple psychiatric disorders and traits A regulator gene with an impact: RBFOX1 and its role in neuropsychiatric disorders Behavioural and functional evidence revealing the role of RBFOX1 variation in multiple psychiatric disorders and traits Global C2orf82 (Snorc) deletion results in distinct behavioural alterations in mice The translational genetics of ADHD and related phenotypes in model organisms Hippocampal overexpression of NOS1AP promotes endophenotypes related to mental disorders Psycho-social factors associated with mental resilience in the Corona lockdown The evolution of dystonia-like movements in TOR1A rats after transient nerve injury is accompanied by dopaminergic dysregulation and abnormal oscillatory activity of a central motor network Defective synaptic transmission causes disease signs in a mouse model of juvenile neuronal ceroid lipofuscinosis Expression of the ADHD candidate gene Diras2 in the brain Prenatal and postnatal experiences associated with epigenetic changes in the adult mouse brain Defective synaptic transmission causes disease signs in a mouse model of Juvenile Neuronal Ceroid Lipofuscinosis Tor1a+/- mice develop dystonia-like movements via a striatal dopaminergic dysregulation triggered by peripheral nerve injury Tor1a+/- mice develop dystonia-like movements via a striatal dopaminergic dysregulation triggered by peripheral nerve injury Tor1a+/- mice develop dystonia-like movements via a striatal dopaminergic dysregulation triggered by peripheral nerve injury

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Katalog der Deutschen Nationalbibliothek

Aet O'Leary (1974-)

A systematic review of preclinical studies exploring the role of insulin signalling in executive function and memory

A systematic review of preclinical studies exploring the role of insulin signalling in executive function and memory

Insulin and disorders of behavioural flexibility

Behavioural and functional evidence revealing the role of RBFOX1 variation in multiple psychiatric disorders and traits

A regulator gene with an impact: RBFOX1 and its role in neuropsychiatric disorders

Behavioural and functional evidence revealing the role of RBFOX1 variation in multiple psychiatric disorders and traits

Global C2orf82 (Snorc) deletion results in distinct behavioural alterations in mice

The translational genetics of ADHD and related phenotypes in model organisms

Hippocampal overexpression of NOS1AP promotes endophenotypes related to mental disorders

Psycho-social factors associated with mental resilience in the Corona lockdown

The evolution of dystonia-like movements in TOR1A rats after transient nerve injury is accompanied by dopaminergic dysregulation and abnormal oscillatory activity of a central motor network

Defective synaptic transmission causes disease signs in a mouse model of juvenile neuronal ceroid lipofuscinosis

Expression of the ADHD candidate gene Diras2 in the brain

Prenatal and postnatal experiences associated with epigenetic changes in the adult mouse brain

Defective synaptic transmission causes disease signs in a mouse model of Juvenile Neuronal Ceroid Lipofuscinosis

Tor1a+/- mice develop dystonia-like movements via a striatal dopaminergic dysregulation triggered by peripheral nerve injury

Tor1a+/- mice develop dystonia-like movements via a striatal dopaminergic dysregulation triggered by peripheral nerve injury

Tor1a+/- mice develop dystonia-like movements via a striatal dopaminergic dysregulation triggered by peripheral nerve injury