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Christian Brendel (1977-)

Biologe

Name	Christian Brendel
Time	1977- (Date of birth and death)
Place	Mainz (Geburtsort) Deutschland
Profession	Biologe (characteristic profession)
Further information	Dissertation Fachbereich Biologie, Univ. Frankfurt am Main
Record type	Person
GND-Identifier	1017426821
Record ID	1017426821
Involved in show all Genetic reversal of the globin switch concurrently modulates both fetal and sickle hemoglobin and reduces red cell sickling Development of lentiviral vectors for the gene therapy of X-linked chronic granulomatous disease Highly efficient therapeutic gene editing of human hematopoietic stem cells A minimal ubiquitous chromatin opening element (UCOE) effectively prevents silencing of juxtaposed heterologous promoters by epigenetic remodeling in multipotent and pluripotent stem cells Stat5 exerts distinct, vital functions in the cytoplasm and nucleus of Bcr-Abl+ K562 and Jak2(V617F)+ HEL leukemia cells From bench to bedside : preclinical evaluation of a self-inactivating gammaretroviral vector for the gene therapy of X-linked chronic granulomatous disease The inhibition of Stat5 by a peptide aptamer ligand specific for the DNA binding domain prevents target gene transactivation and the growth of breast and prostate tumor cells Development of lentiviral vectors for the gene therapy of X-linked chronic granulomatous disease Development of lentiviral vectors for the gene therapy of X-linked chronic granulomatous disease

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Christian Brendel (1977-)

Genetic reversal of the globin switch concurrently modulates both fetal and sickle hemoglobin and reduces red cell sickling

Development of lentiviral vectors for the gene therapy of X-linked chronic granulomatous disease

Highly efficient therapeutic gene editing of human hematopoietic stem cells

A minimal ubiquitous chromatin opening element (UCOE) effectively prevents silencing of juxtaposed heterologous promoters by epigenetic remodeling in multipotent and pluripotent stem cells

Stat5 exerts distinct, vital functions in the cytoplasm and nucleus of Bcr-Abl+ K562 and Jak2(V617F)+ HEL leukemia cells

From bench to bedside : preclinical evaluation of a self-inactivating gammaretroviral vector for the gene therapy of X-linked chronic granulomatous disease

The inhibition of Stat5 by a peptide aptamer ligand specific for the DNA binding domain prevents target gene transactivation and the growth of breast and prostate tumor cells

Development of lentiviral vectors for the gene therapy of X-linked chronic granulomatous disease

Development of lentiviral vectors for the gene therapy of X-linked chronic granulomatous disease