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Jasmin Wellbrock

Biologin

Name	Jasmin Wellbrock
Place	Deutschland
Gender	weiblich
Academic degree	Dr. rer. nat., Dipl.-Biol.
Profession	Biologin (characteristic profession) Postdoc
Variant	Jasmin Otten (früherer Name) Jasmin Wellbrock-Otten (vollständiger Name) Jasmin Wellbrock- Otten (vollständiger Name)
Related	Universitätsklinikum Hamburg-Eppendorf
Further information	Univ. Hamburg, Medizinische Fak., Universitätsklinikum Hamburg-Eppendorf, Onkologisches Zentrum, II. Medizinische Klinik und Poliklinik, Arbeitsgruppe Angiogenese
Record type	Person
GND-Identifier	102822656X
Record ID	102822656X
Involved in show all Bedeutung der GLI-Transkriptionsfaktoren in der Resistenzentwicklung gegenüber Chemotherapie und deren Inhibition in der akuten myeloischen Leukämie Funktionelle Bedeutung der E-Cadherin-Fragmente für die Metastasierung des Ovarialkarzinoms The Actin Binding Protein Plastin-3 Is Involved in the Pathogenesis of Acute Myeloid Leukemia The role of the guanine nucleotide exchange factors DOCK2 and DOCK5 in acute myeloid leukemia and their interactions with epigenetic regulation The BET bromodomain inhibitor ZEN-3365 targets the Hedgehog signaling pathway in acute myeloid leukemia Mebendazole Mediates Proteasomal Degradation of GLI Transcription Factors in Acute Myeloid Leukemia Interaction of PVR/PVRL2 with TIGIT/DNAM-1 as a novel immune checkpoint axis and therapeutic target in cancer Cortisol facilitates the immune escape of human acute myeloid leukemia cells by inducing latrophilin 1 expression Immune checkpoints PVR and PVRL2 are prognostic markers in AML and their blockade represents a new therapeutic option

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Jasmin Wellbrock

Bedeutung der GLI-Transkriptionsfaktoren in der Resistenzentwicklung gegenüber Chemotherapie und deren Inhibition in der akuten myeloischen Leukämie

Funktionelle Bedeutung der E-Cadherin-Fragmente für die Metastasierung des Ovarialkarzinoms

The Actin Binding Protein Plastin-3 Is Involved in the Pathogenesis of Acute Myeloid Leukemia

The role of the guanine nucleotide exchange factors DOCK2 and DOCK5 in acute myeloid leukemia and their interactions with epigenetic regulation

The BET bromodomain inhibitor ZEN-3365 targets the Hedgehog signaling pathway in acute myeloid leukemia

Mebendazole Mediates Proteasomal Degradation of GLI Transcription Factors in Acute Myeloid Leukemia

Interaction of PVR/PVRL2 with TIGIT/DNAM-1 as a novel immune checkpoint axis and therapeutic target in cancer

Cortisol facilitates the immune escape of human acute myeloid leukemia cells by inducing latrophilin 1 expression

Immune checkpoints PVR and PVRL2 are prognostic markers in AML and their blockade represents a new therapeutic option