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Person

Sabine Jägle (1981-)

Biologin

Name	Sabine Jägle
Time	1981- (Date of birth and death)
Place	Zell am Harmersbach (Geburtsort) Deutschland
Profession	Biologin (characteristic profession)
Related	Molekularbiologie (Studienfach)
Further information	Diss. 2014, Universität Freiburg, Fakultät für Biologie
Record type	Person
GND-Identifier	1054304262
Record ID	1054304262
Involved in show all A distinct CD38+CD45RA+ population of CD4+, CD8+, and double-negative T cells is controlled by FAS Genotype–phenotype correlation in von Hippel‐Lindau disease Marfan syndrome caused by disruption of the FBN1 gene due to a reciprocal chromosome translocation Porokeratosis plantaris, palmaris et disseminata caused by con- genital pathogenic variants in the MVD gene and loss of heterozygosity in affected skin A family with palmar and plantar hyperkeratosis: a quiz SNAIL1-mediated downregulation of FOXA proteins facilitates the inactivation of transcriptional enhancer elements at key epithelial genes in colorectal cancer cells The role of a transcriptional enhancer in silencing of the EPHB3 tumor-suppressor gene in colorectal cancer cells Acetylation of human TCF4 (TCF7L2) proteins attenuates inhibition by the HBP1 repressor and induces a conformational change in the TCF4::DNA complex

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Sabine Jägle (1981-)

A distinct CD38+CD45RA+ population of CD4+, CD8+, and double-negative T cells is controlled by FAS

Genotype–phenotype correlation in von Hippel‐Lindau disease

Marfan syndrome caused by disruption of the FBN1 gene due to a reciprocal chromosome translocation

Porokeratosis plantaris, palmaris et disseminata caused by con- genital pathogenic variants in the MVD gene and loss of hetero­zygosity in affected skin

A family with palmar and plantar hyperkeratosis: a quiz

SNAIL1-mediated downregulation of FOXA proteins facilitates the inactivation of transcriptional enhancer elements at key epithelial genes in colorectal cancer cells

The role of a transcriptional enhancer in silencing of the EPHB3 tumor-suppressor gene in colorectal cancer cells

Acetylation of human TCF4 (TCF7L2) proteins attenuates inhibition by the HBP1 repressor and induces a conformational change in the TCF4::DNA complex

Porokeratosis plantaris, palmaris et disseminata caused by con- genital pathogenic variants in the MVD gene and loss of heterozygosity in affected skin