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Dominik Christian Fuhrmann (1985-)

Name	Dominik Christian Fuhrmann
Time	1985- (Date of birth and death)
Place	Frankfurt am Main (Geburtsort) Deutschland
Gender	männlich
Academic degree	Dr. (am Fachbereich Medizin der Universität Frankfurt (Main)) (2015)
Variant	Dominik Fuhrmann D. C. Fuhrmann
Record type	Person
GND-Identifier	1071604619
Record ID	1071604619
Involved in show all Identification of a novel form of caspase-independent cell death triggered by BH3-mimetics in diffuse large B-cell lymphoma cell lines Tumor associated macrophages transfer ceruloplasmin mRNA to fibrosarcoma cells and protect them from ferroptosis HIF1α-AS1 is a DNA:DNA:RNA triplex-forming lncRNA interacting with the HUSH complex miR-193a-3p increases glycolysis under hypoxia by facilitating Akt phosphorylation and PFKFB3 activation in human macrophages A graphical journey through iron metabolism, microRNAs, and hypoxia in ferroptosis Effects of hypoxia on RNA cargo in extracellular vesicles from human adipose-derived stromal/stem cells ER-stress promotes VHL-independent degradation of hypoxia-inducible factors via FBXW1A/βTrCP Functional RNA dynamics are progressively governed by RNA destabilization during the adaptation to chronic hypoxia Identification of the cysteine protease legumain as a potential chronic hypoxia-specific multiple myeloma target gene MicroRNA-200c attenuates the tumor-infiltrating capacity of macrophages miR-193a-3p increases glycolysis under hypoxia by facilitating Akt phosphorylation and PFKFB3 activation in human macrophages Characterization of extracellular vesicles from preconditioned human adipose-derived stromal/stem cells Lactate dehydrogenase B regulates macrophage metabolism in the tumor microenvironment Reduced exploratory behavior in neuronal nucleoredoxin knockout mice Hypoxia inhibits ferritinophagy, increases mitochondrial ferritin, and protects from ferroptosis Metabolic plasticity is an essential requirement of acquired tyrosine kinase inhibitor resistance in chronic myeloid leukemia Chronic hypoxia enhances β-oxidation-dependent electron transport via electron transferring flavoproteins Strategies to interfere with tumor metabolism through the interplay of innate and adaptive immunity Polarization of human macrophages by interleukin-4 does not require ATP-citrate lyase Mitochondrial composition and function under the control of hypoxia Loss of HIF-1α in macrophages attenuates AhR/ARNT-mediated tumorigenesis in a PAH-driven tumor model Chronic hypoxia in human macrophages : adaption and functional consequences

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Dominik Christian Fuhrmann (1985-)

Identification of a novel form of caspase-independent cell death triggered by BH3-mimetics in diffuse large B-cell lymphoma cell lines

Tumor associated macrophages transfer ceruloplasmin mRNA to fibrosarcoma cells and protect them from ferroptosis

HIF1α-AS1 is a DNA:DNA:RNA triplex-forming lncRNA interacting with the HUSH complex

miR-193a-3p increases glycolysis under hypoxia by facilitating Akt phosphorylation and PFKFB3 activation in human macrophages

A graphical journey through iron metabolism, microRNAs, and hypoxia in ferroptosis

Effects of hypoxia on RNA cargo in extracellular vesicles from human adipose-derived stromal/stem cells

ER-stress promotes VHL-independent degradation of hypoxia-inducible factors via FBXW1A/βTrCP

Functional RNA dynamics are progressively governed by RNA destabilization during the adaptation to chronic hypoxia

Identification of the cysteine protease legumain as a potential chronic hypoxia-specific multiple myeloma target gene

MicroRNA-200c attenuates the tumor-infiltrating capacity of macrophages

miR-193a-3p increases glycolysis under hypoxia by facilitating Akt phosphorylation and PFKFB3 activation in human macrophages

Characterization of extracellular vesicles from preconditioned human adipose-derived stromal/stem cells

Lactate dehydrogenase B regulates macrophage metabolism in the tumor microenvironment

Reduced exploratory behavior in neuronal nucleoredoxin knockout mice

Hypoxia inhibits ferritinophagy, increases mitochondrial ferritin, and protects from ferroptosis

Metabolic plasticity is an essential requirement of acquired tyrosine kinase inhibitor resistance in chronic myeloid leukemia

Chronic hypoxia enhances β-oxidation-dependent electron transport via electron transferring flavoproteins

Strategies to interfere with tumor metabolism through the interplay of innate and adaptive immunity

Polarization of human macrophages by interleukin-4 does not require ATP-citrate lyase

Mitochondrial composition and function under the control of hypoxia

Loss of HIF-1α in macrophages attenuates AhR/ARNT-mediated tumorigenesis in a PAH-driven tumor model

Chronic hypoxia in human macrophages : adaption and functional consequences