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Theresa Tretter

Name	Theresa Tretter
Place	Heidelberg (Wirkungsort) Deutschland
Gender	weiblich
Academic degree	Dr. rer. nat.
Variant	T. Tretter
Related	Universitätsklinikum Heidelberg
Further information	Wissenschaftliche Mitarbeiterin, Med. Klinik der Universität Heidelberg, Innere Medizin, Abt. Hämatologie, Onkologie u. Rheumatologie
Record type	Person
GND-Identifier	1083287559
Record ID	1083287559
Involved in show all Third-generation CD19.CAR-T cell-containing combination therapy in Scl70+ systemic sclerosis JAK inhibition prevents the induction of pro-inflammatory HLA-DR+CD90+ RA synovial fibroblasts by IFN[gamma] Increase of aerobic glycolysis mediated by activated T helper cells drives synovial fibroblasts towards an inflammatory phenotype: new targets for therapy? Proinflammatory T cell polarization is already present in patients with early knee osteoarthritis Activation of natural killer cells by rituximab in granulomatosis with polyangiitis Peripheral blood natural killer cell percentages in granulomatosis with polyangiitis correlate with disease inactivity and stage CD4+CD25+/highCD127low/- regulatory T cells are enriched in rheumatoid arthritis and osteoarthritis joints—analysis of frequency and phenotype in synovial membrane, synovial fluid and peripheral blood

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Theresa Tretter

Third-generation CD19.CAR-T cell-containing combination therapy in Scl70+ systemic sclerosis

JAK inhibition prevents the induction of pro-inflammatory HLA-DR+CD90+ RA synovial fibroblasts by IFN[gamma]

Increase of aerobic glycolysis mediated by activated T helper cells drives synovial fibroblasts towards an inflammatory phenotype: new targets for therapy?

Proinflammatory T cell polarization is already present in patients with early knee osteoarthritis

Activation of natural killer cells by rituximab in granulomatosis with polyangiitis

Peripheral blood natural killer cell percentages in granulomatosis with polyangiitis correlate with disease inactivity and stage

CD4+CD25+/highCD127low/- regulatory T cells are enriched in rheumatoid arthritis and osteoarthritis joints—analysis of frequency and phenotype in synovial membrane, synovial fluid and peripheral blood