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Aladin Haimovici (1984-)

Name	Aladin Haimovici
Time	1984- (Date of birth and death)
Place	Schweiz
Gender	männlich
Variant	Aladin-Khaled Haimovici
Related	Biologie (Studienfach)
Record type	Person
GND-Identifier	108400884X
Record ID	108400884X
Involved in show all The Caspase-Activated DNase drives inflammation and contributes to defense against viral infection The Caspase-Activated DNase drives inflammation and contributes to defense against viral infection The caspase-activated DNase promotes cellular senescence Sub-lethal signals in the mitochondrial apoptosis apparatus: pernicious by-product or physiological event? Sub-lethal signals in the mitochondrial apoptosis apparatus: pernicious by-product or physiological event? Correction to: Mitochondria supply sub-lethal signals for cytokine secretion and DNA-damage in H. pylori infection Mitochondria supply sub-lethal signals for cytokine secretion and DNA-damage in H. pylori infection Correction to: Mitochondria supply sub-lethal signals for cytokine secretion and DNA-damage in H. pylori infection Mitochondria supply sub-lethal signals for cytokine secretion and DNA-damage in H. pylori infection Spontaneous activity of the mitochondrial apoptosis pathway drives chromosomal defects, the appearance of micronuclei and cancer metastasis through the Caspase-Activated DNAse A non‐death function of the mitochondrial apoptosis apparatus in immunity Inhibitor of apoptosis proteins are required for effective fusion of autophagosomes with lysosomes Inhibitor of apoptosis proteins are required for effective fusion of autophagosomes with lysosomes PU.1 supports TRAIL-induced cell death by inhibiting NF-κB-mediated cell survival and inducing DR5 expression

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Aladin Haimovici (1984-)

The Caspase-Activated DNase drives inflammation and contributes to defense against viral infection

The Caspase-Activated DNase drives inflammation and contributes to defense against viral infection

The caspase-activated DNase promotes cellular senescence

Sub-lethal signals in the mitochondrial apoptosis apparatus: pernicious by-product or physiological event?

Sub-lethal signals in the mitochondrial apoptosis apparatus: pernicious by-product or physiological event?

Correction to: Mitochondria supply sub-lethal signals for cytokine secretion and DNA-damage in H. pylori infection

Mitochondria supply sub-lethal signals for cytokine secretion and DNA-damage in H. pylori infection

Correction to: Mitochondria supply sub-lethal signals for cytokine secretion and DNA-damage in H. pylori infection

Mitochondria supply sub-lethal signals for cytokine secretion and DNA-damage in H. pylori infection

Spontaneous activity of the mitochondrial apoptosis pathway drives chromosomal defects, the appearance of micronuclei and cancer metastasis through the Caspase-Activated DNAse

A non‐death function of the mitochondrial apoptosis apparatus in immunity

Inhibitor of apoptosis proteins are required for effective fusion of autophagosomes with lysosomes

Inhibitor of apoptosis proteins are required for effective fusion of autophagosomes with lysosomes

PU.1 supports TRAIL-induced cell death by inhibiting NF-κB-mediated cell survival and inducing DR5 expression