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Person

Thomas Zeyen (1994-)

Arzt

Name	Thomas Zeyen
Time	1994- (Date of birth and death)
Place	Düren Deutschland
Academic degree	Dr. med.
Profession	Arzt (characteristic profession)
Variant	T. Zeyen Z. Thomas
Further information	Dissertation an der Rheinisch-Westfälischen Technischen Hochschule Aachen, Medizinische Fakultät, 2020
Source	Dissertation
Record type	Person
GND-Identifier	1223931099
Record ID	1223931099
Involved in show all Correction to: Erythropoietin abrogates post-ischemic activation of the NLRP3, NLRC4, and AIM2inflammasomes in microglia/macrophages in a TAK1-dependent manner Correction to: Erythropoietin Abrogates Post-Ischemic Activation of the NLRP3, NLRC4, and AIM2 Inflammasomes in Microglia/Macrophages in a TAK1-Dependent Manner Correction to: Erythropoietin Abrogates Post-Ischemic Activation of the NLRP3, NLRC4, and AIM2 Inflammasomes in Microglia/Macrophages in a TAK1-Dependent Manner Phase I/II trial of meclofenamate in progressive MGMT-methylated glioblastoma under temozolomide second-line therapy—the MecMeth/NOA-24 trial Erythropoietin abrogates post-ischemic activation of the NLRP3, NLRC4, and AIM2 inflammasomes in microglia/macrophages in a TAK1-dependent manner Phase I/II trial of meclofenamate in progressive MGMT-methylated glioblastoma under temozolomide second-line therapy - the MecMeth/NOA-24 trial Phase I/II trial of meclofenamate in progressive MGMT-methylated glioblastoma under temozolomide second-line therapy-the MecMeth/NOA-24 trial Correction to: Erythropoietin Abrogates Post-ischemic Activation of the NLRP3, NLRC4, and AIM2 Inflammasomes in Microglia/Macrophages in a TAK1-Dependent Manner Erythropoietin Abrogates Post-Ischemic Activation of the NLRP3, NLRC4, and AIM2 Inflammasomes in Microglia/Macrophages in a TAK1-Dependent Manner Microglial-specific depletion of TAK1 is neuroprotective in the acute phase after ischemic stroke Microglial-specific depletion of TAK1 is neuroprotective in the acute phase after ischemic stroke Microglial-specific depletion of TAK1 is neuroprotective in the acute phase after ischemic stroke

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Thomas Zeyen (1994-)

Correction to: Erythropoietin abrogates post-ischemic activation of the NLRP3, NLRC4, and AIM2inflammasomes in microglia/macrophages in a TAK1-dependent manner

Correction to: Erythropoietin Abrogates Post-Ischemic Activation of the NLRP3, NLRC4, and AIM2 Inflammasomes in Microglia/Macrophages in a TAK1-Dependent Manner

Correction to: Erythropoietin Abrogates Post-Ischemic Activation of the NLRP3, NLRC4, and AIM2 Inflammasomes in Microglia/Macrophages in a TAK1-Dependent Manner

Phase I/II trial of meclofenamate in progressive MGMT-methylated glioblastoma under temozolomide second-line therapy—the MecMeth/NOA-24 trial

Erythropoietin abrogates post-ischemic activation of the NLRP3, NLRC4, and AIM2 inflammasomes in microglia/macrophages in a TAK1-dependent manner

Phase I/II trial of meclofenamate in progressive MGMT-methylated glioblastoma under temozolomide second-line therapy - the MecMeth/NOA-24 trial

Phase I/II trial of meclofenamate in progressive MGMT-methylated glioblastoma under temozolomide second-line therapy-the MecMeth/NOA-24 trial

Correction to: Erythropoietin Abrogates Post-ischemic Activation of the NLRP3, NLRC4, and AIM2 Inflammasomes in Microglia/Macrophages in a TAK1-Dependent Manner

Erythropoietin Abrogates Post-Ischemic Activation of the NLRP3, NLRC4, and AIM2 Inflammasomes in Microglia/Macrophages in a TAK1-Dependent Manner

Microglial-specific depletion of TAK1 is neuroprotective in the acute phase after ischemic stroke

Microglial-specific depletion of TAK1 is neuroprotective in the acute phase after ischemic stroke

Microglial-specific depletion of TAK1 is neuroprotective in the acute phase after ischemic stroke