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Martin Hasselblatt (1969-)

Arzt

Name	Martin Hasselblatt
Time	1969- (Date of birth and death)
Place	Münster (Westf) (Wirkungsort) Deutschland
Academic degree	Prof- Dr. med.
Profession	Arzt (characteristic profession)
Related	Westfälische Wilhelms-Universität Münster. Medizinische Fakultät
Further information	Diss. Medizinische Fakultät; Institut für Neuropathologie, Medizinische Fakultät Münster
Record type	Person
GND-Identifier	123559014
Record ID	123559014
Involved in show all EpiDiP/NanoDiP: a versatile unsupervised machine learning edge computing platform for epigenomic tumour diagnostics Radiation therapy plays an important role in the treatment of atypical teratoid/rhabdoid tumors: analysis of the EU-RHAB cohorts and their precursors Author Correction: Multiomic neuropathology improves diagnostic accuracy in pediatric neuro-oncology Recurrent atypical teratoid/rhabdoid tumors (AT/RT) reveal discrete features of progression on histology, epigenetics, copy number profiling, and transcriptomics Multiomic neuropathology improves diagnostic accuracy in pediatric neuro-oncology Current molecular and clinical landscape of ATRT: the link to future therapies DNA methylation profiling of central nervous system hemangioblastomas identifies two distinct subgroups Multiomic neuropathology improves diagnostic accuracy in pediatric neuro-oncology Primary cilia contribute to the aggressiveness of atypical teratoid/rhabdoid tumors ATRT–SHH comprises three molecular subgroups with characteristic clinical and histopathological features and prognostic significance DNA methylation-based classification of sinonasal tumors Infants and Newborns with Atypical Teratoid Rhabdoid Tumors (ATRT) and Extracranial Malignant Rhabdoid Tumors (eMRT) in the EU-RHAB Registry : A Unique and Challenging Population Infants and Newborns with Atypical Teratoid Rhabdoid Tumors (ATRT) and Extracranial Malignant Rhabdoid Tumors (eMRT) in the EU-RHAB Registry: A Unique and Challenging Population Low-grade diffusely infiltrative tumour (LGDIT), SMARCB1-mutant: A clinical and histopathological distinct entity showing epigenetic similarity with ATRT-MYC Low‐grade diffusely infiltrative tumour (LGDIT), SMARCB1‐mutant: a clinical and histopathological distinct entity showing epigenetic similarity with ATRT‐MYC Genetic and epigenetic characterization of posterior pituitary tumors GOPC:ROS1 and other ROS1 fusions represent a rare but recurrent drug target in a variety of glioma types Inhibition of nuclear export restores nuclear localization and residual tumor suppressor function of truncated SMARCB1/INI1 protein in a molecular subset of atypical teratoid/rhabdoid tumors Glioblastomas with primitive neuronal component harbor a distinct methylation and copy-number profile with inactivation of TP53, PTEN, and RB1 Glioblastomas with primitive neuronal component harbor a distinct methylation and copy-number profile with inactivation of TP53, PTEN, and RB1 TERT promoter mutation and chromosome 6 loss define a high-risk subtype of ependymoma evolving from posterior fossa subependymoma Clinical evidence for a biological effect of epigenetically active decitabine in relapsed or progressive rhabdoid tumors Clinical evidence for a biological effect of epigenetically active decitabine in relapsed or progressive rhabdoid tumors Clinical evidence for a biological effect of epigenetically active decitabine in relapsed or progressive rhabdoid tumors Histopathological patterns in atypical teratoid/rhabdoid tumors are related to molecular subgroup Transposable element insertion as a mechanism of SMARCB1 inactivation in atypical teratoid/rhabdoid tumor Atypical teratoid/rhabdoid tumors (ATRTs) with SMARCA4 mutation are molecularly distinct from SMARCB1-deficient cases Clear cell meningiomas are defined by a highly distinct DNA methylation profile and mutations in SMARCE1 Primary mismatch repair deficient IDH-mutant astrocytoma (PMMRDIA) is a distinct type with a poor prognosis Molecular characterization of CNS paragangliomas identifies cauda equina paragangliomas as a distinct tumor entity Epigenetics impacts upon prognosis and clinical management of choroid plexus tumors Atypical teratoid/rhabdoid tumors (ATRTs) with SMARCA4 mutation are molecularly distinct from SMARCB1-deficient cases Improved 6-year overall survival in AT/RT - results of the registry study Rhabdoid 2007 Macrophage-tumor cell interaction promotes ATRT progression and chemoresistance Molecular subgrouping of primary pineal parenchymal tumors reveals distinct subtypes correlated with clinical parameters and genetic alterations Desmoplastic myxoid tumor, SMARCB1-mutant: clinical, histopathological and molecular characterization of a pineal region tumor encountered in adolescents and adults Posterior fossa pilocytic astrocytomas with oligodendroglial features show frequent FGFR1 activation via fusion or mutation Molecular characterization of histopathological ependymoma variants Correction to: DNA methylation signature is prognostic of choroid plexus tumor aggressiveness Isomorphic diffuse glioma is a morphologically and molecularly distinct tumour entity with recurrent gene fusions of MYBL1 or MYB and a benign disease course DNA methylation signature is prognostic of choroid plexus tumor aggressiveness Rosette-forming glioneuronal tumors share a distinct DNA methylation profile and mutations in FGFR1, with recurrent co-mutation of PIK3CA and NF1 Two molecularly distinct atypical teratoid/rhabdoid tumors (or tumor components) occurring in an infant with rhabdoid tumor predisposition syndrome 1 Identification and analyses of extra-cranial and cranial rhabdoid tumor molecular subgroups reveal tumors with cytotoxic T cell infiltration Molecular subgrouping of atypical teratoid/rhabdoid tumors: a reinvestigation and current consensus Functional relevance of genes predicted to be affected by epigenetic alterations in atypical teratoid/rhabdoid tumors FGFR1:TACC1 fusion is a frequent event in molecularly defined extraventricular neurocytoma Germline variants in SMARCB1 and other members of the BAF chromatin-remodeling complex across human disease entities: a meta-analysis Characterization of Diffuse Gliomas With Histone H3-G34 Mutation by MRI and Dynamic 18F-FET PET SWI/SNF-Komplex-assoziierte Tumordispositions-Syndrome Reduced histone H3 K27 trimethylation is encountered in about 50% of atypical teratoid/rhabdoid tumors (AT/RT) but is not associated with molecular subgroup status and outcome Abstracts of the 52nd Workshop for Pediatric Research : Frankfurt, Germany. 27-28 October 2016 SMAD dependent signaling plays a detrimental role in a fly model of SMARCB1-deficiency and the biology of atypical teratoid/rhabdoid tumors hTERT promoter methylation in meningiomas and central nervous hemangiopericytomas Poorly differentiated chordoma with SMARCB1/INI1 loss: a distinct molecular entity with dismal prognosis Germline and somatic FGFR1 abnormalities in dysembryoplastic neuroepithelial tumors Improved 6-year overall survival in AT/RT - results of the registry study rhabdoid 2007 New brain tumor entities emerge from molecular classification of CNS-PNETs Histologically distinct neuroepithelial tumors with histone 3 G34 mutation are molecularly similar and comprise a single nosologic entity Pediatric atypical choroid plexus papilloma reconsidered: increased mitotic activity is prognostic only in older children

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Martin Hasselblatt (1969-)

EpiDiP/NanoDiP: a versatile unsupervised machine learning edge computing platform for epigenomic tumour diagnostics

Radiation therapy plays an important role in the treatment of atypical teratoid/rhabdoid tumors: analysis of the EU-RHAB cohorts and their precursors

Author Correction: Multiomic neuropathology improves diagnostic accuracy in pediatric neuro-oncology

Recurrent atypical teratoid/rhabdoid tumors (AT/RT) reveal discrete features of progression on histology, epigenetics, copy number profiling, and transcriptomics

Multiomic neuropathology improves diagnostic accuracy in pediatric neuro-oncology

Current molecular and clinical landscape of ATRT: the link to future therapies

DNA methylation profiling of central nervous system hemangioblastomas identifies two distinct subgroups

Multiomic neuropathology improves diagnostic accuracy in pediatric neuro-oncology

Primary cilia contribute to the aggressiveness of atypical teratoid/rhabdoid tumors

ATRT–SHH comprises three molecular subgroups with characteristic clinical and histopathological features and prognostic significance

DNA methylation-based classification of sinonasal tumors

Infants and Newborns with Atypical Teratoid Rhabdoid Tumors (ATRT) and Extracranial Malignant Rhabdoid Tumors (eMRT) in the EU-RHAB Registry : A Unique and Challenging Population

Infants and Newborns with Atypical Teratoid Rhabdoid Tumors (ATRT) and Extracranial Malignant Rhabdoid Tumors (eMRT) in the EU-RHAB Registry: A Unique and Challenging Population

Low-grade diffusely infiltrative tumour (LGDIT), SMARCB1-mutant: A clinical and histopathological distinct entity showing epigenetic similarity with ATRT-MYC

Low‐grade diffusely infiltrative tumour (LGDIT), SMARCB1‐mutant: a clinical and histopathological distinct entity showing epigenetic similarity with ATRT‐MYC

Genetic and epigenetic characterization of posterior pituitary tumors

GOPC:ROS1 and other ROS1 fusions represent a rare but recurrent drug target in a variety of glioma types

Inhibition of nuclear export restores nuclear localization and residual tumor suppressor function of truncated SMARCB1/INI1 protein in a molecular subset of atypical teratoid/rhabdoid tumors

Glioblastomas with primitive neuronal component harbor a distinct methylation and copy-number profile with inactivation of TP53, PTEN, and RB1

Glioblastomas with primitive neuronal component harbor a distinct methylation and copy-number profile with inactivation of TP53, PTEN, and RB1

TERT promoter mutation and chromosome 6 loss define a high-risk subtype of ependymoma evolving from posterior fossa subependymoma

Clinical evidence for a biological effect of epigenetically active decitabine in relapsed or progressive rhabdoid tumors

Clinical evidence for a biological effect of epigenetically active decitabine in relapsed or progressive rhabdoid tumors

Clinical evidence for a biological effect of epigenetically active decitabine in relapsed or progressive rhabdoid tumors

Histopathological patterns in atypical teratoid/rhabdoid tumors are related to molecular subgroup

Transposable element insertion as a mechanism of SMARCB1 inactivation in atypical teratoid/rhabdoid tumor

Atypical teratoid/rhabdoid tumors (ATRTs) with SMARCA4 mutation are molecularly distinct from SMARCB1-deficient cases

Clear cell meningiomas are defined by a highly distinct DNA methylation profile and mutations in SMARCE1

Primary mismatch repair deficient IDH-mutant astrocytoma (PMMRDIA) is a distinct type with a poor prognosis

Molecular characterization of CNS paragangliomas identifies cauda equina paragangliomas as a distinct tumor entity

Epigenetics impacts upon prognosis and clinical management of choroid plexus tumors

Atypical teratoid/rhabdoid tumors (ATRTs) with SMARCA4 mutation are molecularly distinct from SMARCB1-deficient cases

Improved 6-year overall survival in AT/RT - results of the registry study Rhabdoid 2007

Macrophage-tumor cell interaction promotes ATRT progression and chemoresistance

Molecular subgrouping of primary pineal parenchymal tumors reveals distinct subtypes correlated with clinical parameters and genetic alterations

Desmoplastic myxoid tumor, SMARCB1-mutant: clinical, histopathological and molecular characterization of a pineal region tumor encountered in adolescents and adults

Posterior fossa pilocytic astrocytomas with oligodendroglial features show frequent FGFR1 activation via fusion or mutation

Molecular characterization of histopathological ependymoma variants

Correction to: DNA methylation signature is prognostic of choroid plexus tumor aggressiveness

Isomorphic diffuse glioma is a morphologically and molecularly distinct tumour entity with recurrent gene fusions of MYBL1 or MYB and a benign disease course

DNA methylation signature is prognostic of choroid plexus tumor aggressiveness

Rosette-forming glioneuronal tumors share a distinct DNA methylation profile and mutations in FGFR1, with recurrent co-mutation of PIK3CA and NF1

Two molecularly distinct atypical teratoid/rhabdoid tumors (or tumor components) occurring in an infant with rhabdoid tumor predisposition syndrome 1

Identification and analyses of extra-cranial and cranial rhabdoid tumor molecular subgroups reveal tumors with cytotoxic T cell infiltration

Molecular subgrouping of atypical teratoid/rhabdoid tumors: a reinvestigation and current consensus

Functional relevance of genes predicted to be affected by epigenetic alterations in atypical teratoid/rhabdoid tumors

FGFR1:TACC1 fusion is a frequent event in molecularly defined extraventricular neurocytoma

Germline variants in SMARCB1 and other members of the BAF chromatin-remodeling complex across human disease entities: a meta-analysis

Characterization of Diffuse Gliomas With Histone H3-G34 Mutation by MRI and Dynamic 18F-FET PET

SWI/SNF-Komplex-assoziierte Tumordispositions-Syndrome

Reduced histone H3 K27 trimethylation is encountered in about 50% of atypical teratoid/rhabdoid tumors (AT/RT) but is not associated with molecular subgroup status and outcome

Abstracts of the 52nd Workshop for Pediatric Research : Frankfurt, Germany. 27-28 October 2016

SMAD dependent signaling plays a detrimental role in a fly model of SMARCB1-deficiency and the biology of atypical teratoid/rhabdoid tumors

hTERT promoter methylation in meningiomas and central nervous hemangiopericytomas

Poorly differentiated chordoma with SMARCB1/INI1 loss: a distinct molecular entity with dismal prognosis

Germline and somatic FGFR1 abnormalities in dysembryoplastic neuroepithelial tumors

Improved 6-year overall survival in AT/RT - results of the registry study rhabdoid 2007

New brain tumor entities emerge from molecular classification of CNS-PNETs

Histologically distinct neuroepithelial tumors with histone 3 G34 mutation are molecularly similar and comprise a single nosologic entity

Pediatric atypical choroid plexus papilloma reconsidered: increased mitotic activity is prognostic only in older children