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Alexander Streuer (1994-)

Assistenzarzt

Name	Alexander Streuer
Time	1994- (Date of birth and death)
Place	Zavodoukovsk (Geburtsort) Deutschland
Gender	männlich
Academic degree	Dr. med. (an der Medizinischen Fakultät Mannheim der Universität Heidelberg) (2022)
Profession	Assistenzarzt (characteristic profession)
Related	Universität Heidelberg. Medizinische Fakultät Mannheim
Further information	Assistenzarzt in der III. Medizinischen Klinik der Medizinischen Fakultät Mannheim der Universität Heidelberg
Record type	Person
GND-Identifier	1237642302
Record ID	1237642302
Involved in show all Targeting tumoral heterogeneity in lung cancer: a novel, CT-texture-guided targeted biopsy approach with exome sequencing The bispecific innate cell engager AFM28 eliminates CD123+ leukemic stem and progenitor cells in AML and MDS Machine learning-based in-silico analysis identifies signatures of lysyl oxidases for prognostic and therapeutic response prediction in cancer Treatment with the apoptosis inhibitor Asunercept reduces clone sizes in patients with lower risk Myelodysplastic Neoplasms Significant improvement of bone marrow-derived MSC expansion from MDS patients by defined xeno-free medium Identifikation neuer molekularer, therapeutischer Zielfaktoren in der Pathogenese von Frühtodesfällen bei der Akuten Promyelozyten Leukämie

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Katalog der Deutschen Nationalbibliothek

Alexander Streuer (1994-)

Targeting tumoral heterogeneity in lung cancer: a novel, CT-texture-guided targeted biopsy approach with exome sequencing

The bispecific innate cell engager AFM28 eliminates CD123+ leukemic stem and progenitor cells in AML and MDS

Machine learning-based in-silico analysis identifies signatures of lysyl oxidases for prognostic and therapeutic response prediction in cancer

Treatment with the apoptosis inhibitor Asunercept reduces clone sizes in patients with lower risk Myelodysplastic Neoplasms

Significant improvement of bone marrow-derived MSC expansion from MDS patients by defined xeno-free medium

Identifikation neuer molekularer, therapeutischer Zielfaktoren in der Pathogenese von Frühtodesfällen bei der Akuten Promyelozyten Leukämie