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Person

Julian P. Maier (1995-)

Arzt

Name	Julian P. Maier
Time	1995- (Date of birth and death)
Place	Gengenbach (Geburtsort) Deutschland
Gender	männlich
Academic degree	Dr. med.
Profession	Arzt (characteristic profession)
Variant	Julian Philipp Maier (vollständiger Name) J. P. Maier Julian Maier J. Maier
Further information	Dissertation 2023, Albert-Ludwigs-Universität Freiburg im Breisgau, Medizinische Fakultät
Source	FreiDok plus AM
Record type	Person
GND-Identifier	1254513868
Record ID	1254513868
Involved in show all Next-generation sequencing of the human aqueous humour microbiome Transcriptional profiling specifies the pathogen-specific human host response to infectious keratitis Glutamate signaling in glioblastoma: Inhibition of metabotropic glutamate receptor III facilitates sensitization to alkylating chemotherapeutics Indications, techniques, and graft survival of mini and corneo-scleral tectonic keratoplasties: a retrospective single-center case series T-cell dysfunction in the glioblastoma microenvironment is mediated by myeloid cells releasing interleukin-10 T-cell dysfunction in the glioblastoma microenvironment is mediated by myeloid cells releasing interleukin-10 Inhibition of metabotropic glutamate receptor III facilitates sensitization to alkylating chemotherapeutics in glioblastoma Astrogliosis releases pro-oncogenic chitinase 3-like 1 causing MAPK signaling in glioblastoma Astrogliosis Releases Pro-Oncogenic Chitinase 3-Like 1 Causing MAPK Signaling in Glioblastoma

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Katalog der Deutschen Nationalbibliothek

Julian P. Maier (1995-)

Next-generation sequencing of the human aqueous humour microbiome

Transcriptional profiling specifies the pathogen-specific human host response to infectious keratitis

Glutamate signaling in glioblastoma: Inhibition of metabotropic glutamate receptor III facilitates sensitization to alkylating chemotherapeutics

Indications, techniques, and graft survival of mini and corneo-scleral tectonic keratoplasties: a retrospective single-center case series

T-cell dysfunction in the glioblastoma microenvironment is mediated by myeloid cells releasing interleukin-10

T-cell dysfunction in the glioblastoma microenvironment is mediated by myeloid cells releasing interleukin-10

Inhibition of metabotropic glutamate receptor III facilitates sensitization to alkylating chemotherapeutics in glioblastoma

Astrogliosis releases pro-oncogenic chitinase 3-like 1 causing MAPK signaling in glioblastoma

Astrogliosis Releases Pro-Oncogenic Chitinase 3-Like 1 Causing MAPK Signaling in Glioblastoma