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Christoph Welsch (1974-)

Name	Christoph Welsch
Time	1974- (Date of birth and death)
Place	Saarlouis (Geburtsort) Deutschland
Academic degree	Dr. med. (Medizinische Fakultät an der Universität Saarbrücken) (2005) Dr. med. habil., Priv.-Doz. (Fachbereich Medizin der Johann Wolfgang Goethe-Universität Frankfurt am Main) (2016)
Record type	Person
GND-Identifier	131851500
Record ID	131851500
Involved in show all Author Correction: Commensal bacteria weaken the intestinal barrier by suppressing epithelial neuropilin-1 and Hedgehog signaling Commensal bacteria weaken the intestinal barrier by suppressing epithelial neuropilin-1 and Hedgehog signaling Differential lipidomics, metabolomics and immunological analysis of alcoholic and non-alcoholic steatohepatitis in mice Distinct clinical phenotypes in a family with a novel truncating MEN1 frameshift mutation Pulmonary impairment independently determines mortality in critically ill patients with acute-on-chronic liver failure Regulatory role of phospholipids in hepatitis C virus replication and protein function Epistatic interactions promote persistence of NS3-Q80K in HCV infection by compensating for protein folding instability Extended interaction networks with HCV protease NS3-4A substrates explain the lack of adaptive capability against protease inhibitors Translation of IRF-1 restricts hepatic interleukin-7 production to types I and II interferons: implications for hepatic immunity Trends and the course of liver cirrhosis and its complications in Germany: Nationwide population-based study (2005 to 2018) Anemia and systemic inflammation rather than arterial circulatory dysfunction predict decompensation of liver cirrhosis The role of macrophage-inducible c-type lectin in different stages of chronic liver disease Macrophage-derived extracellular vesicles induce long-lasting immunity against Hepatitis C virus which is blunted by polyunsaturated fatty acids Macrophage-derived extracellular vesicles induce long-lasting immunity against Hepatitis C virus which is blunted by Polyunsaturated fatty acids Molecular basis of telaprevir resistance due to V36 and T54 mutations in the NS3-4A protease of the hepatitis C virus Molecular basis of telaprevir resistance due to V36 and T54 mutations in the NS3-4A protease of the hepatitis C virus ESR-spektroskopische Charakterisierung von NO-Targets in humanem Blutplasma

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Christoph Welsch (1974-)

Author Correction: Commensal bacteria weaken the intestinal barrier by suppressing epithelial neuropilin-1 and Hedgehog signaling

Commensal bacteria weaken the intestinal barrier by suppressing epithelial neuropilin-1 and Hedgehog signaling

Differential lipidomics, metabolomics and immunological analysis of alcoholic and non-alcoholic steatohepatitis in mice

Distinct clinical phenotypes in a family with a novel truncating MEN1 frameshift mutation

Pulmonary impairment independently determines mortality in critically ill patients with acute-on-chronic liver failure

Regulatory role of phospholipids in hepatitis C virus replication and protein function

Epistatic interactions promote persistence of NS3-Q80K in HCV infection by compensating for protein folding instability

Extended interaction networks with HCV protease NS3-4A substrates explain the lack of adaptive capability against protease inhibitors

Translation of IRF-1 restricts hepatic interleukin-7 production to types I and II interferons: implications for hepatic immunity

Trends and the course of liver cirrhosis and its complications in Germany: Nationwide population-based study (2005 to 2018)

Anemia and systemic inflammation rather than arterial circulatory dysfunction predict decompensation of liver cirrhosis

The role of macrophage-inducible c-type lectin in different stages of chronic liver disease

Macrophage-derived extracellular vesicles induce long-lasting immunity against Hepatitis C virus which is blunted by polyunsaturated fatty acids

Macrophage-derived extracellular vesicles induce long-lasting immunity against Hepatitis C virus which is blunted by Polyunsaturated fatty acids

Molecular basis of telaprevir resistance due to V36 and T54 mutations in the NS3-4A protease of the hepatitis C virus

Molecular basis of telaprevir resistance due to V36 and T54 mutations in the NS3-4A protease of the hepatitis C virus

ESR-spektroskopische Charakterisierung von NO-Targets in humanem Blutplasma