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Peter Claus

Anatom

Name	Peter Claus
Place	Hannover (Wirkungsort) Deutschland
Gender	männlich
Academic degree	Prof. Dr. rer. nat.
Profession	Anatom (characteristic profession)
Related	Medizinische Hochschule Hannover Medizinische Hochschule Hannover. Institut für Neuroanatomie
Further information	Habil. 2005; Diss. Göttingen; PD für Anatomie
Record type	Person
GND-Identifier	132297248
Record ID	132297248
Involved in show all Morphologic examination of upper respiratory tract epithelium with special consideration of olfactory epithelium after SARS-CoV-2 infection Actin-binding proteins in Spinal Muscular Atrophy (SMA) and other neurodegenerative diseases : Molecular disease mechanisms POLE, POLD1 und CDH1 als Gliomprädispositionsgene : Identifizierung von seltenen tumorassoziierten Keimbahnvarianten in Gliomfamilien und deren Charakterisierung durch Untersuchung von Tumoren von Variantenträgern und Zellmodellen Transcriptomic profiling of the hippocampus and frontal cortex in the context of cognitive dysfunction in mouse models of Spinal Muscular Atrophy and Parkinson's disease In vitro investigation of a canine histiocytic sarcoma cell line following an infection with genetically modified and non-modified canine distemper virus strains In vitro investigation of a canine histiocytic sarcoma cell line following an infection with genetically modified and non-modified canine distemper virus strains Phenotypic characteristics of satellite glial cells of sensory ganglia with special reference to species-specific variations and commonalities Application of Antisense Oligonucleotides and Adenoassociated Virus in a Spinal Muscular Atrophy Model Modulation of innate immune responses within the central nervous system in a mouse model of neurotropic virus infection Molecular mechanisms and dysregulated networks in Amyotrophic Lateral Sclerosis and Spinal Muscular Atrophy Dynamics and regulation of the actin cytoskeleton in the motoneuron disease Spinal Muscular Atrophy (SMA) Neuronal cultures from dorsal root ganglia of adult dogs represent a promising in vitro model to investigate infectious and non-infectious influences Biochemische Charakterisierung von Ena/VASP- und Formin-Aktinpolymerasen Modulation Aktin-regulierender Signalkaskaden in einem Modell der Spinalen Muskelatrophobie (SMA)

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Peter Claus

Morphologic examination of upper respiratory tract epithelium with special consideration of olfactory epithelium after SARS-CoV-2 infection

Actin-binding proteins in Spinal Muscular Atrophy (SMA) and other neurodegenerative diseases : Molecular disease mechanisms

POLE, POLD1 und CDH1 als Gliomprädispositionsgene : Identifizierung von seltenen tumorassoziierten Keimbahnvarianten in Gliomfamilien und deren Charakterisierung durch Untersuchung von Tumoren von Variantenträgern und Zellmodellen

Transcriptomic profiling of the hippocampus and frontal cortex in the context of cognitive dysfunction in mouse models of Spinal Muscular Atrophy and Parkinson's disease

In vitro investigation of a canine histiocytic sarcoma cell line following an infection with genetically modified and non-modified canine distemper virus strains

In vitro investigation of a canine histiocytic sarcoma cell line following an infection with genetically modified and non-modified canine distemper virus strains

Phenotypic characteristics of satellite glial cells of sensory ganglia with special reference to species-specific variations and commonalities

Application of Antisense Oligonucleotides and Adenoassociated Virus in a Spinal Muscular Atrophy Model

Modulation of innate immune responses within the central nervous system in a mouse model of neurotropic virus infection

Molecular mechanisms and dysregulated networks in Amyotrophic Lateral Sclerosis and Spinal Muscular Atrophy

Dynamics and regulation of the actin cytoskeleton in the motoneuron disease Spinal Muscular Atrophy (SMA)

Neuronal cultures from dorsal root ganglia of adult dogs represent a promising in vitro model to investigate infectious and non-infectious influences

Biochemische Charakterisierung von Ena/VASP- und Formin-Aktinpolymerasen

Modulation Aktin-regulierender Signalkaskaden in einem Modell der Spinalen Muskelatrophobie (SMA)