Detail page

Knowledge Web

Person

Martin Eilers (1960-)

Biochemiker

Name	Martin Eilers
Time	1960- (Date of birth and death)
Place	Deutschland
Profession	Biochemiker (characteristic profession)
Related	Julius-Maximilians-Universität Würzburg (Professor für Biochemie und Molekularbiologie) Deutsche Akademie der Naturforscher Leopoldina (Mitglied) (2015-)
Topic	Personen zu allgemeiner Biologie, Mikrobiologie Personen zu Chemie
Source	LCAuth
Record type	Person
GND-Identifier	141366443
Record ID	141366443
Involved in show all PAF1c links S-phase progression to immune evasion and MYC function in pancreatic carcinoma Targeting MYC effector functions in pancreatic cancer by inhibiting the ATPase RUVBL1/2 LRP8‐mediated selenocysteine uptake is a targetable vulnerability in MYCN‐amplified neuroblastoma MYC promotes immune-suppression in triple-negative breast cancer via inhibition of interferon signaling Publisher Correction: Pharmacological reactivation of MYC-dependent apoptosis induces susceptibility to anti-PD-1 immunotherapy Pharmacological reactivation of MYC-dependent apoptosis induces susceptibility to anti-PD-1 immunotherapy Protein kinase D1 deletion in adipocytes enhances energy dissipation and protects against adiposity MYC and tumor metabolism: chicken and egg The MYC mRNA 3′‐UTR couples RNA polymerase II function to glutamine and ribonucleotide levels The first sino-german symposium on DNA repair and human diseases

Please note

The beta version does not yet contain all functions and information of the DNB portal catalogue. If you are missing information or want to order a medium, please visit the page in the DNB portal catalogue via the following link:

Katalog der Deutschen Nationalbibliothek

Martin Eilers (1960-)

PAF1c links S-phase progression to immune evasion and MYC function in pancreatic carcinoma

Targeting MYC effector functions in pancreatic cancer by inhibiting the ATPase RUVBL1/2

LRP8‐mediated selenocysteine uptake is a targetable vulnerability in MYCN‐amplified neuroblastoma

MYC promotes immune-suppression in triple-negative breast cancer via inhibition of interferon signaling

Publisher Correction: Pharmacological reactivation of MYC-dependent apoptosis induces susceptibility to anti-PD-1 immunotherapy

Pharmacological reactivation of MYC-dependent apoptosis induces susceptibility to anti-PD-1 immunotherapy

Protein kinase D1 deletion in adipocytes enhances energy dissipation and protects against adiposity

MYC and tumor metabolism: chicken and egg

The MYC mRNA 3′‐UTR couples RNA polymerase II function to glutamine and ribonucleotide levels

The first sino-german symposium on DNA repair and human diseases