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David Reuss (1976-)

Arzt

Name	David Reuss
Time	1976- (Date of birth and death)
Place	Berlin (Geburtsort) Heidelberg (Wirkungsort) Deutschland
Academic degree	Dr. med. (Universität Heidelberg)
Profession	Arzt (characteristic profession) Privatdozent
Variant	David Reuß David Emanuel Reuss David E. Reuss David E. Reuß
Related	Universität Heidelberg. Pathologisches Institut (seit 2008)
Further information	Ltd. Oberarzt der Abteilung Neuropathologie
Record type	Person
GND-Identifier	143839837
Record ID	143839837
Involved in show all Genetical and epigenetical profiling identifies two subgroups of pineal parenchymal tumors of intermediate differentiation (PPTID) with distinct molecular, histological and clinical characteristics Single-cell DNA sequencing reveals order of mutational acquisition in TRAF7/AKT1 and TRAF7/KLF4 mutant meningiomas Molecular features of glioblastomas in long-term survivors compared to short-term survivors—a matched-pair analysis Pleomorphic xanthoastrocytoma is a heterogeneous entity with pTERT mutations prognosticating shorter survival Drug sensitivity profiling of 3D tumor tissue cultures in the pediatric precision oncology program INFORM Molecular features of glioblastomas in long-term survivors compared to short-term survivors—a matched-pair analysis Molecular matched targeted therapies for primary brain tumors - a single center retrospective analysis Oligosarcomas, IDH-mutant are distinct and aggressive Oligosarcomas, IDH-mutant are distinct and aggressive Recurrent fusions in PLAGL1 define a distinct subset of pediatric-type supratentorial neuroepithelial tumors Glioblastomas with primitive neuronal component harbor a distinct methylation and copy-number profile with inactivation of TP53, PTEN, and RB1 Glioblastomas with primitive neuronal component harbor a distinct methylation and copy-number profile with inactivation of TP53, PTEN, and RB1 Molecular analysis of pediatric CNS-PNET revealed nosologic heterogeneity and potent diagnostic markers for CNS neuroblastoma with FOXR2-activation Recurrent fusions in PLAGL1 define a distinct subset of pediatric-type supratentorial neuroepithelial tumors Clear cell meningiomas are defined by a highly distinct DNA methylation profile and mutations in SMARCE1 Primary mismatch repair deficient IDH-mutant astrocytoma (PMMRDIA) is a distinct type with a poor prognosis Infratentorial IDH-mutant astrocytoma is a distinct subtype CDKN2A/B homozygous deletion is associated with early recurrence in meningiomas YAP1-fusions in pediatric NF2-wildtype meningioma YAP1-fusions in pediatric NF2-wildtype meningioma Posterior fossa pilocytic astrocytomas with oligodendroglial features show frequent FGFR1 activation via fusion or mutation Tumors diagnosed as cerebellar glioblastoma comprise distinct molecular entities Isomorphic diffuse glioma is a morphologically and molecularly distinct tumour entity with recurrent gene fusions of MYBL1 or MYB and a benign disease course MYCN amplification drives an aggressive form of spinal ependymoma Routine RNA sequencing of formalin-fixed paraffin-embedded specimens in neuropathology diagnostics identifies diagnostically and therapeutically relevant gene fusions Rosette-forming glioneuronal tumors share a distinct DNA methylation profile and mutations in FGFR1, with recurrent co-mutation of PIK3CA and NF1 Mutational patterns and regulatory networks in epigenetic subgroups of meningioma Papillary glioneuronal tumor (PGNT) exhibits a characteristic methylation profile and fusions involving PRKCA High frequency of H3 K27M mutations in adult midline gliomas Genome-wide methylation profiling and copy number analysis in atypical fibroxanthomas and pleomorphic dermal sarcomas indicate a similar molecular phenotype Mutational patterns and regulatory networks in epigenetic subgroups of meningioma Heterogeneity of glioblastoma with gliomatosis cerebri growth pattern on diffusion and perfusion MRI The molecular landscape of glioma in patients with Neurofibromatosis 1 Chordoid meningiomas can be sub-stratified into prognostically distinct DNA methylation classes and are enriched for heterozygous deletions of chromosomal arm 2p Distribution of EGFR amplification, combined chromosome 7 gain and chromosome 10 loss, and TERT promoter mutation in brain tumors and their potential for the reclassification of IDHwt astrocytoma to glioblastoma FGFR1:TACC1 fusion is a frequent event in molecularly defined extraventricular neurocytoma Practical implementation of DNA methylation and copy-number-based CNS tumor diagnostics: the Heidelberg experience Primary intracranial spindle cell sarcoma with rhabdomyosarcoma-like features share a highly distinct methylation profile and DICER1 mutations Novel, improved grading system(s) for IDH-mutant astrocytic gliomas Array-based DNA-methylation profiling in sarcomas with small blue round cell histology provides valuable diagnostic information Anaplastic astrocytoma with piloid features, a novel molecular class of IDH wildtype glioma with recurrent MAPK pathway, CDKN2A/B and ATRX alterations Rapid detection of 2-hydroxyglutarate in frozen sections of IDH mutant tumors by MALDI-TOF mass spectrometry Proteomics in Pathology Identification of T cell target antigens in glioblastoma stem-like cells using an integrated proteomics-based approach in patient specimens Gain of 12p encompassing CCND2 is associated with gemistocytic histology in IDH mutant astrocytomas Methylation-based classification of benign and malignant peripheral nerve sheath tumors Next-generation sequencing in routine brain tumor diagnostics enables an integrated diagnosis and identifies actionable targets Histologically distinct neuroepithelial tumors with histone 3 G34 mutation are molecularly similar and comprise a single nosologic entity Adult IDH wild type astrocytomas biologically and clinically resolve into other tumor entities Neurofibromin hemmt das Tumorzellwachstum durch RasGAP-unabhängige Suppression des Klasse II Transaktivators

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Katalog der Deutschen Nationalbibliothek

David Reuss (1976-)

Genetical and epigenetical profiling identifies two subgroups of pineal parenchymal tumors of intermediate differentiation (PPTID) with distinct molecular, histological and clinical characteristics

Single-cell DNA sequencing reveals order of mutational acquisition in TRAF7/AKT1 and TRAF7/KLF4 mutant meningiomas

Molecular features of glioblastomas in long-term survivors compared to short-term survivors—a matched-pair analysis

Pleomorphic xanthoastrocytoma is a heterogeneous entity with pTERT mutations prognosticating shorter survival

Drug sensitivity profiling of 3D tumor tissue cultures in the pediatric precision oncology program INFORM

Molecular features of glioblastomas in long-term survivors compared to short-term survivors—a matched-pair analysis

Molecular matched targeted therapies for primary brain tumors - a single center retrospective analysis

Oligosarcomas, IDH-mutant are distinct and aggressive

Oligosarcomas, IDH-mutant are distinct and aggressive

Recurrent fusions in PLAGL1 define a distinct subset of pediatric-type supratentorial neuroepithelial tumors

Glioblastomas with primitive neuronal component harbor a distinct methylation and copy-number profile with inactivation of TP53, PTEN, and RB1

Glioblastomas with primitive neuronal component harbor a distinct methylation and copy-number profile with inactivation of TP53, PTEN, and RB1

Molecular analysis of pediatric CNS-PNET revealed nosologic heterogeneity and potent diagnostic markers for CNS neuroblastoma with FOXR2-activation

Recurrent fusions in PLAGL1 define a distinct subset of pediatric-type supratentorial neuroepithelial tumors

Clear cell meningiomas are defined by a highly distinct DNA methylation profile and mutations in SMARCE1

Primary mismatch repair deficient IDH-mutant astrocytoma (PMMRDIA) is a distinct type with a poor prognosis

Infratentorial IDH-mutant astrocytoma is a distinct subtype

CDKN2A/B homozygous deletion is associated with early recurrence in meningiomas

YAP1-fusions in pediatric NF2-wildtype meningioma

YAP1-fusions in pediatric NF2-wildtype meningioma

Posterior fossa pilocytic astrocytomas with oligodendroglial features show frequent FGFR1 activation via fusion or mutation

Tumors diagnosed as cerebellar glioblastoma comprise distinct molecular entities

Isomorphic diffuse glioma is a morphologically and molecularly distinct tumour entity with recurrent gene fusions of MYBL1 or MYB and a benign disease course

MYCN amplification drives an aggressive form of spinal ependymoma

Routine RNA sequencing of formalin-fixed paraffin-embedded specimens in neuropathology diagnostics identifies diagnostically and therapeutically relevant gene fusions

Rosette-forming glioneuronal tumors share a distinct DNA methylation profile and mutations in FGFR1, with recurrent co-mutation of PIK3CA and NF1

Mutational patterns and regulatory networks in epigenetic subgroups of meningioma

Papillary glioneuronal tumor (PGNT) exhibits a characteristic methylation profile and fusions involving PRKCA

High frequency of H3 K27M mutations in adult midline gliomas

Genome-wide methylation profiling and copy number analysis in atypical fibroxanthomas and pleomorphic dermal sarcomas indicate a similar molecular phenotype

Mutational patterns and regulatory networks in epigenetic subgroups of meningioma

Heterogeneity of glioblastoma with gliomatosis cerebri growth pattern on diffusion and perfusion MRI

The molecular landscape of glioma in patients with Neurofibromatosis 1

Chordoid meningiomas can be sub-stratified into prognostically distinct DNA methylation classes and are enriched for heterozygous deletions of chromosomal arm 2p

Distribution of EGFR amplification, combined chromosome 7 gain and chromosome 10 loss, and TERT promoter mutation in brain tumors and their potential for the reclassification of IDHwt astrocytoma to glioblastoma

FGFR1:TACC1 fusion is a frequent event in molecularly defined extraventricular neurocytoma

Practical implementation of DNA methylation and copy-number-based CNS tumor diagnostics: the Heidelberg experience

Primary intracranial spindle cell sarcoma with rhabdomyosarcoma-like features share a highly distinct methylation profile and DICER1 mutations

Novel, improved grading system(s) for IDH-mutant astrocytic gliomas

Array-based DNA-methylation profiling in sarcomas with small blue round cell histology provides valuable diagnostic information

Anaplastic astrocytoma with piloid features, a novel molecular class of IDH wildtype glioma with recurrent MAPK pathway, CDKN2A/B and ATRX alterations

Rapid detection of 2-hydroxyglutarate in frozen sections of IDH mutant tumors by MALDI-TOF mass spectrometry

Proteomics in Pathology

Identification of T cell target antigens in glioblastoma stem-like cells using an integrated proteomics-based approach in patient specimens

Gain of 12p encompassing CCND2 is associated with gemistocytic histology in IDH mutant astrocytomas

Methylation-based classification of benign and malignant peripheral nerve sheath tumors

Next-generation sequencing in routine brain tumor diagnostics enables an integrated diagnosis and identifies actionable targets

Histologically distinct neuroepithelial tumors with histone 3 G34 mutation are molecularly similar and comprise a single nosologic entity

Adult IDH wild type astrocytomas biologically and clinically resolve into other tumor entities

Neurofibromin hemmt das Tumorzellwachstum durch RasGAP-unabhängige Suppression des Klasse II Transaktivators